项目编号 PRJCA011606
项目标题 Knocking out BATF improves CAR-T cell efficacy by resisting exhaustion and forming central memory cells
涉及领域 Medical
先导项目

eGPS: evolutionary Genotype-Phenotype Systems biology

Plateau Adaptability

Analysis of adaptive traits in the Qinghai Tibet Plateau

数据类型 Exome
物种名称 Mus musculus
Homo sapiens
描述信息 Chimeric antigen receptor modified T (CAR-T) cell therapy has limited efficacy against solid tumor, one major challenge is T cell exhaustion. To address this challenge, we performed a candidate gene screen using a hypofunction CAR-T cell model, and found that knocking out BATF improved the performance of CAR-T cells. In different types of CAR-T cells and mouse OT-1 cells, knocking out BATF endows T cells with improved resistance to exhaustion and better tumor eradication efficacy. We find that BATF binds to and up-regulates a subset of exhaustion genes in human CAR-T cells. Furthermore, BATF regulates the expression of genes involved in the development of effector and memory cells, and knocking out BATF shifts the population towards more central memory subset. Therefore, we conclude that BATF is a key factor limiting CAR-T cell function, and its depletion improves CAR-T cells efficacy against solid tumor.
样品范围 CAR-T cells
发布日期 2022-09-01
出版信息
PubMed ID 文章标题 杂志名称 Doi 发表年份
36240777 Depletion of BATF in CAR-T cells enhances antitumor activity by inducing resistance against exhaustion and formation of central memory cells Cancer Cell 10.1016/j.ccell.2022.09.013 2022
项目资金来源
机构 项目类型 授权项目ID 授权项目名称
Chinese Academy of Sciences (CAS) N/A
提交者 Xingying Zhang (zhangxingying@ioz.ac.cn)
提交单位 Institute of Zoology, Chinese Academy of Sciences
提交日期 2022-09-01

项目包含数据信息

资源名称 描述
BioSample (40)  show -
GSA (1) -
CRA008037 Knocking out BATF improves CAR-T cell efficacy by resisting exhaustion and forming central memory cells