RiceWiki - User contributions [en]

AB462324

2014-06-09T02:13:35Z

Zhangshoudong: /* Structured Information */

==Annotated Information==
===Function===
The recently cloned blast resistance (R) gene Pi-km protects rice crops against specific races of the fungal pathogen Magnaporthe oryzae in a gene-for-gene manner.[1]High-resolution genetic mapping and sequencing of the gene region in the Pikm-containing cultivar Tsuyuake narrowed down the candidate region to a 131-kb genomic interval[2]
====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake
====基因的发现与命名====
Kiyosawa 报道Minehikari 中含有一个与Pi-k 连锁的抗性基因Pi-m，但后来多个试验表明Pi-m 与Pi-k 等位，于是将Pi-m 重新命名为Pi-km (Kiyosawa, 1978)。

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===

===Evolution===

==Labs working on this gene==
Please input related labs here.
==References==
1. Stefano Costanzo;Yulin Jia,Sequence variation at the rice blast resistance gene Pi-km locus: Implications for the development of allele specific markers.Plant Science, 2010, 178(6): 523-530

2.Ikuo Ashikawa;Nagao Hayashi;Hiroko Yamane;Hiroyuki Kanamori;Jianzhong Wu;Takashi Matsumoto;Kazuko Ono;Masahiro Yano, Two Adjacent Nucleotide-Binding Site–Leucine-Rich Repeat Class Genes Are Required to Confer Pikm-Specific Rice Blast Resistance.Genetics, 2008, 180(4): 2267-2276

3. Luo-Ye Li;Ling Wang;Jin-Xue Jing;Zhen-Qi Li;Fei Lin;Li-Fei Huang;Qing-Hua Pan,The Pikm gene, conferring stable resistance to isolates of Magnaporthe oryzae, was finely mapped in a crossover-cold region on rice chromosome 11. Molecular Breeding, 2007, 20(2): 179-188

4. Shigehisa Kiyosawa,Identification of Blast-Resistance Genes in Some Rice Varieties.Japanese Journal of Breeding, 1978, 28(4): 287-296
==Structured Information==
{{JaponicaGene|
GeneName = AB462325|
Description = 3458 bp mRNA linear PLN 20-MAR-2009.|
Version = AB462325.1 GI:207367331|
Length = 3458 bp|
Definition = Oryza sativa Japonica Group Pikm2-TS mRNA for NBS-LRR class disease
resistance protein, complete cds.|
Source = Oryza sativa Japonica Group (Japanese rice)
ORGANISM Oryza sativa Japonica Group
Eukaryota; Viridiplantae; Streptophyta; Embryophyta; Tracheophyta;
Spermatophyta; Magnoliophyta; Liliopsida; Poales; Poaceae; BEP
clade; Ehrhartoideae; Oryzeae; Oryza.
|
Chromosome = [[:category:Japonica Chromosome 11|Chromosome 11]]|
AP = Chromosome11:110..3175|
CDS =110..3175|
AA = <aaseq>MELVVGASEATMKSLLGKLGNLLAQEYALISGIRGDIQYINDEL
ASMQAFLRDLSNVPEGHSHGHRMKDWMKQIRDIAYDVEDCIDDFAHRLPQDSISDAKW
SFLLTKIYELWTWWPRRVIASNIAQLKVRAQQIADRRSRYGVNNPEHLDSSSSARTRA
VNYEIAEYQVTSPQIIGIKEPVGMKTVMEELEVWLTNPQAENGQAVLSIVGFGGVGKT
TIATALYRKVSEKFQCRASVAVSQNYDQGKVLNSILSQVSNQEQGSSTTISEKKNLTS
GAKSMLKTALSLLRGNCICQPENDGNPDNTPIRLQETTDDDQNPRKLEQLLAEKSYIL
LIDDIWSAETWESIRSILPKNNKGGRIIVTTRFQAVGSTCSPLETDRLHTVDFLTDDE
SQNLFNTSICESKIRKDSNKVDEQVPEEIWKICGGLPLAIVSMAGLVACNPRKACCDW
SKLCKSLFPEQETPLTLDGVTRILDCCYNDLPADLKTCLLYLSIFPKGWKISRKRLSR
RWIAEGFANEKQGLTQERVAEAYFNQLTRRNLVRPMEHGSNGKVKTFQVHDMVLEYIM
SKSIEENFITVVGGHWQMTAPSNKVRRLSMQSSGSNRGSSTKGLNLAQVRSLTVFGNL
NHVPFHSFNYGIIQVLDLEDWKGLKERHMTEICQMLLLKYLSIRRTEISKIPSKIQKL
EYLETLDIRETYVRDLPKSIVQLKRIISILGGNKNTRKGLRLPQEKSKKPIKNPSPQG
KTKEPAKKGFLSQEKGKGAMKALRVLSGIEIVEESSEVAAGLHQLTGLRKLAIYKLNI
TKGGDTFKQLQSSIEYLGSCGLQTLAINDENSEFINSLGDMPAPPRYLVALELSGKLE
KLPKWITSITTLNKLTISVTVLRTETLEILHILPSLFSLTFAFSLSAAKQDQDIIKDI
LENNKLDSDGEIVIPAEGFKSLKLLRFFAPLVPKLSFLDKNAMPALEIIEMRFKDFEG
LFGIEILENLREVHLKVSDGAEAITKFLVNDLKVNTEKPKVFVDGIVTA</aaseq>|
DNA =<dnaseqindica>1..61#121..181#241..301#361..421# 481..541#601..661#721..781#841..901#961..1021#1081..1141#1201..1261#1321..1381#1441..1501#1561..1621#1681..1741#1801..1861#1921..1981#2041..2101#2161..2221#2281..2341#
2401..2461#2521..2581#2641..2701#2761..2821#2881..2941#3001..3061#3121..3181#3241..3301#3361..3421# aggacacgga aattctcagc aggttcgcgc ttgatctgaa ctgtctgtgg ctcatactct cttgtgcttg caacagctga aagttgcagt gagaagtaca gagaacaaga tggagttggt ggtaggtgct tccgaagcca ccatgaaatc tctcttgggc aagctgggca atcttctagc ccaggagtat gctctcatca gcggtatccg tggtgacatc cagtacatca atgacgagct tgccagcatg caggccttcc tccgtgatct cagcaacgtg ccagagggtc acagtcatgg ccaccggatg aaggactgga tgaagcagat ccgagacatc gcctatgatg ttgaggactg tatcgatgac tttgcccacc gcctccctca ggattccatc agcgatgcca aatggtcctt cctactcaca aaaatctatg aactatggac atggtggcca cgtcgtgtga ttgcttccaa cattgcccaa ctcaaggtac gggcacaaca gatcgcagat cgacgtagta gatacggagt gaacaaccca gaacaccttg acagtagcag cagtgccagg acccgtgctg tcaattacga aattgctgag tatcaggtca caagccctca gatcattggt ataaaggagc ctgtggggat gaagacggtc atggaggagc ttgaggtttg gttaactaat cctcaagctg aaaatgggca agctgttctg tccatagtcg gttttggagg tgtgggaaag actaccattg ccacagcatt gtacagaaaa gtcagtgaaa aatttcagtg ccgggcatca gtagctgtgt ctcagaacta tgaccaaggc aaagtcctca atagtattct gagtcaagtc agcaatcagg agcagggcag cagcacaaca attagtgaga aaaagaacct cacctcaggc gctaagagca tgttgaagac agccctgtca ctgctcagag gtaattgtat atgtcagcca gaaaatgatg gaaaccctga
taatacacca atcaggctgc aggaaacaac ggacgatgat caaaacccca gaaaactgga
acagctcctg gccgaaaaga gttatatcct cttgattgat gacatttggt ctgccgaaac
atgggagagt atcagatcga ttttgcctaa aaataataaa ggcggtagaa taatagtgac
tacaagattt caagctgttg gttcaacatg ctcccctctt gaaactgatc gtttgcatac
agttgatttt ctcaccgatg acgagtccca aaacttattc aatacaagta tttgtgaatc
aaagataaga aaagatagca acaaagtaga cgagcaagtc cctgaggaaa tatggaaaat
atgtggggga ttgcctttgg ccatagtcag catggctggt cttgtcgcct gcaacccaag
gaaagcctgc tgcgattgga gtaaactttg caaatcatta tttccagagc aagaaactcc
tcttaccctc gatggtgtta caaggatact ggattgttgt tacaatgatt tgcctgcgga
tctgaagact tgcttattgt acttgagtat atttccgaag ggttggaaaa ttagtaggaa
acgtttgtcc cggcgatgga tagctgaagg ttttgctaat gagaagcaag ggttaaccca
ggaaagagtt gcagaggcat actttaatca actcacaaga aggaacttag tacgtcccat
ggagcatggc agcaatggga aggtaaaaac gtttcaagtt catgacatgg ttcttgaata
catcatgtcc aaatcaatcg aagagaattt tattactgtg gttggtggac actggcagat
gactgcacca agcaataaag tccgtcgact gtcgatgcaa agcagtggat ccaatcgtgg
aagttcaaca aaaggcctga acttggctca agtgagatca ctgacggtgt ttgggaacct
gaaccatgtg ccattccatt cattcaacta tgggataata caggtgctgg atcttgagga
ctggaagggt ttgaaagaga gacatatgac ggagatatgt caaatgcttt tactcaagta
tttgagcatc cgacgaacag aaatttccaa aattccctcc aagattcaga aacttgagta
cttggaaact cttgacataa gggagacata tgtcagggac ctgcctaagt caatagtcca
gctaaaacgg atcattagca tacttggagg gaataaaaac acacggaagg ggctgaggtt
gcctcaagaa aaaagtaaga agccaattaa aaacccgtcg cctcaaggaa aaacaaagga
gcccgcaaag aaaggattct tatcccaaga aaaaggtaaa ggcgcaatga aagcactccg
tgtactgtca gggattgaga ttgttgagga atcatcagaa gtagctgcag gccttcatca
gttgacaggg ctaaggaagc ttgccatata caagctcaat ataacaaagg gtggtgatac
cttcaaacaa ttacagtcct ccattgagta ccttggcagc tgtggtctgc agactctggc
catcaatgat gagaattctg aatttatcaa ctcactgggc gacatgcccg cgcctccaag
atatcttgtc gcccttgagc tgtctggcaa gttggagaag ctacccaagt ggatcaccag
catcactact ctcaacaagc taaccatatc tgtaacagtt cttaggactg aaactttgga
gatcctccac attttacctt cattgttttc cctcaccttc gccttttcac ttagtgcagc
gaagcaggat caggacataa taaaggacat ccttgagaat aataaattgg acagtgatgg
ggaaatcgtc attccagctg aaggattcaa gagtcttaag ctgcttcgct tctttgcacc
tttagtgccg aagctcagct ttttggacaa gaatgcaatg ccagcactcg aaatcattga
aatgcggttt aaagacttcg aaggtctatt tggcatcgaa atccttgaaa atctccgtga
ggtgcatctc aaagttagtg atggggcaga agcaataacc aagttccttg taaatgattt
gaaggttaat actgagaaac caaaagtatt tgttgatggc atcgtcactg catgagaagt
aaaattgctg caaatcggag aacttaccaa tcatctgagg cttcccctct attattactc
tcttagaata tattgttatt attgctcacc ttgcaaaata aaatagggat ggcatagcat
attgctacaa cgtaccatgg ttccatcata gttgatttca cttgtcatta cagtgtctgt
tcagttgtgt tttctattaa taaaagggag atctccgcaa gaaaccatta ttatacttat
attcggttat tgaactctat aaatgatggg attgctat</dnaseqindica>|
Link = [http://www.ncbi.nlm.nih.gov/nuccore/207367331]|
}}
[[Category:Genes]]
[[Category:Oryza Sativa Japonica Group]]
[[Category:Japonica Genes]]
[[Category:Japonica Chromosome 11]]
[[Category:Chromosome 11]]

AB462324

2014-06-09T02:12:56Z

Zhangshoudong: /* Structured Information */

AB462324

2014-06-09T02:06:42Z

Zhangshoudong: /* Structured Information */

AB462324

2014-06-09T01:37:21Z

Zhangshoudong: /* References */

AB462324

2014-06-09T01:36:02Z

Zhangshoudong: /* Evolution */

AB462324

2014-06-09T01:34:42Z

Zhangshoudong: /* Function */

AB462324

2014-06-09T01:31:46Z

Zhangshoudong: /* References */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake
====基因的发现与命名====
Kiyosawa 报道Minehikari 中含有一个与Pi-k 连锁的抗性基因Pi-m，但后来多个试验表明Pi-m 与Pi-k 等位，于是将Pi-m 重新命名为Pi-km (Kiyosawa, 1978)。

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===

===Evolution===
==Labs working on this gene==
Please input related labs here.
==References==
1. Stefano Costanzo;Yulin Jia,Sequence variation at the rice blast resistance gene Pi-km locus: Implications for the development of allele specific markers.Plant Science, 2010, 178(6): 523-530

2.Ikuo Ashikawa;Nagao Hayashi;Hiroko Yamane;Hiroyuki Kanamori;Jianzhong Wu;Takashi Matsumoto;Kazuko Ono;Masahiro Yano, Two Adjacent Nucleotide-Binding Site–Leucine-Rich Repeat Class Genes Are Required to Confer Pikm-Specific Rice Blast Resistance.Genetics, 2008, 180(4): 2267-2276

3. Luo-Ye Li;Ling Wang;Jin-Xue Jing;Zhen-Qi Li;Fei Lin;Li-Fei Huang;Qing-Hua Pan,The Pikm gene, conferring stable resistance to isolates of Magnaporthe oryzae, was finely mapped in a crossover-cold region on rice chromosome 11. Molecular Breeding, 2007, 20(2): 179-188

4. Shigehisa Kiyosawa,Identification of Blast-Resistance Genes in Some Rice Varieties.Japanese Journal of Breeding, 1978, 28(4): 287-296

AB462324

2014-06-09T01:31:10Z

Zhangshoudong: /* References */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake
====基因的发现与命名====
Kiyosawa 报道Minehikari 中含有一个与Pi-k 连锁的抗性基因Pi-m，但后来多个试验表明Pi-m 与Pi-k 等位，于是将Pi-m 重新命名为Pi-km (Kiyosawa, 1978)。

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===

===Evolution===
==Labs working on this gene==
Please input related labs here.
==References==
1. Stefano Costanzo;Yulin Jia,Sequence variation at the rice blast resistance gene Pi-km locus: Implications for the development of allele specific markers.Plant Science, 2010, 178(6): 523-530
2. Ikuo Ashikawa;Nagao Hayashi;Hiroko Yamane;Hiroyuki Kanamori;Jianzhong Wu;Takashi Matsumoto;Kazuko Ono;Masahiro Yano, Two Adjacent Nucleotide-Binding Site–Leucine-Rich Repeat Class Genes Are Required to Confer Pikm-Specific Rice Blast Resistance.Genetics, 2008, 180(4): 2267-2276
3. Luo-Ye Li;Ling Wang;Jin-Xue Jing;Zhen-Qi Li;Fei Lin;Li-Fei Huang;Qing-Hua Pan,The Pikm gene, conferring stable resistance to isolates of Magnaporthe oryzae, was finely mapped in a crossover-cold region on rice chromosome 11. Molecular Breeding, 2007, 20(2): 179-188
4. Shigehisa Kiyosawa,Identification of Blast-Resistance Genes in Some Rice Varieties.Japanese Journal of Breeding, 1978, 28(4): 287-296

AB462324

2014-06-09T01:30:07Z

Zhangshoudong: /* References */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake
====基因的发现与命名====
Kiyosawa 报道Minehikari 中含有一个与Pi-k 连锁的抗性基因Pi-m，但后来多个试验表明Pi-m 与Pi-k 等位，于是将Pi-m 重新命名为Pi-km (Kiyosawa, 1978)。

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===

===Evolution===
==Labs working on this gene==
Please input related labs here.
==References==
1. Stefano Costanzo;Yulin Jia
Sequence variation at the rice blast resistance gene Pi-km locus: Implications for the development of allele specific markers
Plant Science, 2010, 178(6): 523-530
2. Ikuo Ashikawa;Nagao Hayashi;Hiroko Yamane;Hiroyuki Kanamori;Jianzhong Wu;Takashi Matsumoto;Kazuko Ono;Masahiro Yano
Two Adjacent Nucleotide-Binding Site–Leucine-Rich Repeat Class Genes Are Required to Confer Pikm-Specific Rice Blast Resistance
Genetics, 2008, 180(4): 2267-2276
3. Luo-Ye Li;Ling Wang;Jin-Xue Jing;Zhen-Qi Li;Fei Lin;Li-Fei Huang;Qing-Hua Pan
The Pikm gene, conferring stable resistance to isolates of Magnaporthe oryzae, was finely mapped in a crossover-cold region on rice chromosome 11
Molecular Breeding, 2007, 20(2): 179-188
4. Shigehisa Kiyosawa
Identification of Blast-Resistance Genes in Some Rice Varieties
Japanese Journal of Breeding, 1978, 28(4): 287-296

AB462324

2014-06-09T01:29:02Z

Zhangshoudong: /* Function */

AB462324

2014-06-09T01:28:03Z

Zhangshoudong: /* Evolution */

AB462324

2014-06-09T01:26:46Z

Zhangshoudong: /* Evolution */

AB462324

2014-06-09T01:26:28Z

Zhangshoudong: /* Expression */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

AB462324

2014-06-09T01:24:59Z

Zhangshoudong: /* 分子标记辅助选择育种 */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====备注====
NBS-LRR 类抗病蛋白

===Expression===
The Pib gene was isolated by a map-based cloning strategy. The deduced amino acid sequence of the Pib gene product contains a nucleotide binding site (NBS) and leucine-rich repeats (LRRs); thus, Pib is a member of the NBS-LRR class of plant disease resistance genes. Interestingly, a duplication of the kinase 1a, 2 and 3a motifs of the NBS region was found in the N-terminal half of the Pib protein. In addition, eight cysteine residues are clustered in the middle of the LRRs, a feature which has not been reported for other R genes. Pib gene expression was induced upon altered environmental conditions, such as altered temperatures and darkness.【3】
Link = [http://www.ricedata.cn/reference/list/507.htm]|

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

AB462324

2014-06-09T01:22:59Z

Zhangshoudong: /* 基因克隆与生物学功能分析 */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
序列分析和遗传互补实验表明，Pi-km 是由两个紧密连锁的具有独立功能NBS-LRR类基因(Pikm1-TS 和Pikm2-TS)组成。其中，Pikm1-TS 包含有62bp 5'-UTR、3432bp 编码区、2个内含子(119bp, 2769bp)和190bp 3'-UTR；Pikm2-TS 与Pikm1-TS 不同源，包含109bp 5'-UTR、3066bp 编码区、1个内含子(163bp)和283bp 3'-UTR(Ashikawa et al., 2008)。
Pikm1-TS 和Pikm2-TS 的编码产物均是NBS-LRR类抗病蛋白，长度分别为1143aa和1021aa。他们在结构上有些小的差异：Pikm1-TS 氨基端发现有nT基序，属于CC结构，而Pikm2-TS 虽也发现有nT基序，但未发现有CC结构；Pikm1-TS 羧基端有非LRR结构，而Pikm2-TS 没有(Ashikawa et al., 2008)。

====分子标记辅助选择育种====
利用抗稻瘟病基因Pib 自身序列及其等位的感病基因序列建立的分子标记结合运用，可有效地从水稻种质资源中快速而准确地选择出抗稻瘟病基因Pib，并能在分离世代群体中选择出含抗稻瘟病基因Pib 的纯合单株(刘洋等, 2008)。
Link = [http://www.ricedata.cn/gene/list/70.htm]|

===Expression===
The Pib gene was isolated by a map-based cloning strategy. The deduced amino acid sequence of the Pib gene product contains a nucleotide binding site (NBS) and leucine-rich repeats (LRRs); thus, Pib is a member of the NBS-LRR class of plant disease resistance genes. Interestingly, a duplication of the kinase 1a, 2 and 3a motifs of the NBS region was found in the N-terminal half of the Pib protein. In addition, eight cysteine residues are clustered in the middle of the LRRs, a feature which has not been reported for other R genes. Pib gene expression was induced upon altered environmental conditions, such as altered temperatures and darkness.【3】
Link = [http://www.ricedata.cn/reference/list/507.htm]|

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

AB462324

2014-06-09T01:22:22Z

Zhangshoudong: /* 基因的定位 */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake

====基因的定位====
Pi-km 初步定位于水稻第11 染色体长臂近末端区域，SSR 标记RM254 和RM144 之间，遗传距离分别是13.4cM 和1.2cM；通过生物信息学分析，发展新的分子标记，最终精细定位在BAC克隆OSJNBa0036K13 内的84kb区间内(Li et al., 2007)。

====基因克隆与生物学功能分析====
Pib 基因属于"NBS-LRR"类抗病基因，包含有4个内含子(164 bp, 810 bp, 1340 bp, 308 bp)，全长cDNA由306bp 的5'非翻译区(UTR, untranslated regions)、3753bp 的ORF 和229bp 的3'非翻译区等组成。Pib 编码一个由1251 个氨基酸组成的蛋白产物，该产物包含一个核苷酸结合位点(nucleotide binding site, NBS)和17个富亮氨酸重复序列(leucine-rich repeats, LRRs)，其中，氨基端的NBS 区存在激酶1a, 2 和3a 结构域单位，LRRs 中部有8 个成簇的半胱氨酸残基(Wang et al., 1999)。
Pib 基因会因环境条件的变化而诱导调控，如温度、光照等条件的改变都将影响该基因的表达(Wang et al., 1999)。

====分子标记辅助选择育种====
利用抗稻瘟病基因Pib 自身序列及其等位的感病基因序列建立的分子标记结合运用，可有效地从水稻种质资源中快速而准确地选择出抗稻瘟病基因Pib，并能在分离世代群体中选择出含抗稻瘟病基因Pib 的纯合单株(刘洋等, 2008)。
Link = [http://www.ricedata.cn/gene/list/70.htm]|

===Expression===
The Pib gene was isolated by a map-based cloning strategy. The deduced amino acid sequence of the Pib gene product contains a nucleotide binding site (NBS) and leucine-rich repeats (LRRs); thus, Pib is a member of the NBS-LRR class of plant disease resistance genes. Interestingly, a duplication of the kinase 1a, 2 and 3a motifs of the NBS region was found in the N-terminal half of the Pib protein. In addition, eight cysteine residues are clustered in the middle of the LRRs, a feature which has not been reported for other R genes. Pib gene expression was induced upon altered environmental conditions, such as altered temperatures and darkness.【3】
Link = [http://www.ricedata.cn/reference/list/507.htm]|

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

AB462324

2014-06-09T01:21:52Z

Zhangshoudong: /* 常规信息 */

==Annotated Information==
===Function===

====常规信息====
Pik-m 是稻瘟病抗性位点Pik 上的一个主效抗病等位基因，供体是Tsuyuake

====基因的定位====
Pib 基因定位于水稻第2 染色体长臂近末端区域，与RFLP标记RZ123, C379, C2782B 等连锁(Miyamoto et al., 1996; Monna et al., 1997)。
Pib 基因定位于水稻第2 染色体上RFLP 标记S1916 和G7030 之间，遗传距离分别为0.015 cM 和0.045 cM，并与RFLP 标记G7010, G7021 和G7023 共分离(Wang et al., 1999)。对应于日本晴测序图谱的位置(5'-3')在35109965 - 35109117 区间(Rice Genome Annotation Project: TIGR version6)。

====基因克隆与生物学功能分析====
Pib 基因属于"NBS-LRR"类抗病基因，包含有4个内含子(164 bp, 810 bp, 1340 bp, 308 bp)，全长cDNA由306bp 的5'非翻译区(UTR, untranslated regions)、3753bp 的ORF 和229bp 的3'非翻译区等组成。Pib 编码一个由1251 个氨基酸组成的蛋白产物，该产物包含一个核苷酸结合位点(nucleotide binding site, NBS)和17个富亮氨酸重复序列(leucine-rich repeats, LRRs)，其中，氨基端的NBS 区存在激酶1a, 2 和3a 结构域单位，LRRs 中部有8 个成簇的半胱氨酸残基(Wang et al., 1999)。
Pib 基因会因环境条件的变化而诱导调控，如温度、光照等条件的改变都将影响该基因的表达(Wang et al., 1999)。

====分子标记辅助选择育种====
利用抗稻瘟病基因Pib 自身序列及其等位的感病基因序列建立的分子标记结合运用，可有效地从水稻种质资源中快速而准确地选择出抗稻瘟病基因Pib，并能在分离世代群体中选择出含抗稻瘟病基因Pib 的纯合单株(刘洋等, 2008)。
Link = [http://www.ricedata.cn/gene/list/70.htm]|

===Expression===
The Pib gene was isolated by a map-based cloning strategy. The deduced amino acid sequence of the Pib gene product contains a nucleotide binding site (NBS) and leucine-rich repeats (LRRs); thus, Pib is a member of the NBS-LRR class of plant disease resistance genes. Interestingly, a duplication of the kinase 1a, 2 and 3a motifs of the NBS region was found in the N-terminal half of the Pib protein. In addition, eight cysteine residues are clustered in the middle of the LRRs, a feature which has not been reported for other R genes. Pib gene expression was induced upon altered environmental conditions, such as altered temperatures and darkness.【3】
Link = [http://www.ricedata.cn/reference/list/507.htm]|

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

AB462324

2014-06-09T01:20:31Z

Zhangshoudong: Created page with "==Annotated Information== ===Function=== ====常规信息==== Pib ，水稻稻瘟病抗性基因，来自品种"BL1"，对日本大多数稻瘟病菌小种有抗性，对中..."

==Annotated Information==
===Function===

====常规信息====
Pib ，水稻稻瘟病抗性基因，来自品种"BL1"，对日本大多数稻瘟病菌小种有抗性，对中国的菌株ZB13 和ZC15 也表现抗病反应。

====基因的定位====
Pib 基因定位于水稻第2 染色体长臂近末端区域，与RFLP标记RZ123, C379, C2782B 等连锁(Miyamoto et al., 1996; Monna et al., 1997)。
Pib 基因定位于水稻第2 染色体上RFLP 标记S1916 和G7030 之间，遗传距离分别为0.015 cM 和0.045 cM，并与RFLP 标记G7010, G7021 和G7023 共分离(Wang et al., 1999)。对应于日本晴测序图谱的位置(5'-3')在35109965 - 35109117 区间(Rice Genome Annotation Project: TIGR version6)。

====基因克隆与生物学功能分析====
Pib 基因属于"NBS-LRR"类抗病基因，包含有4个内含子(164 bp, 810 bp, 1340 bp, 308 bp)，全长cDNA由306bp 的5'非翻译区(UTR, untranslated regions)、3753bp 的ORF 和229bp 的3'非翻译区等组成。Pib 编码一个由1251 个氨基酸组成的蛋白产物，该产物包含一个核苷酸结合位点(nucleotide binding site, NBS)和17个富亮氨酸重复序列(leucine-rich repeats, LRRs)，其中，氨基端的NBS 区存在激酶1a, 2 和3a 结构域单位，LRRs 中部有8 个成簇的半胱氨酸残基(Wang et al., 1999)。
Pib 基因会因环境条件的变化而诱导调控，如温度、光照等条件的改变都将影响该基因的表达(Wang et al., 1999)。

====分子标记辅助选择育种====
利用抗稻瘟病基因Pib 自身序列及其等位的感病基因序列建立的分子标记结合运用，可有效地从水稻种质资源中快速而准确地选择出抗稻瘟病基因Pib，并能在分离世代群体中选择出含抗稻瘟病基因Pib 的纯合单株(刘洋等, 2008)。
Link = [http://www.ricedata.cn/gene/list/70.htm]|

===Expression===
The Pib gene was isolated by a map-based cloning strategy. The deduced amino acid sequence of the Pib gene product contains a nucleotide binding site (NBS) and leucine-rich repeats (LRRs); thus, Pib is a member of the NBS-LRR class of plant disease resistance genes. Interestingly, a duplication of the kinase 1a, 2 and 3a motifs of the NBS region was found in the N-terminal half of the Pib protein. In addition, eight cysteine residues are clustered in the middle of the LRRs, a feature which has not been reported for other R genes. Pib gene expression was induced upon altered environmental conditions, such as altered temperatures and darkness.【3】
Link = [http://www.ricedata.cn/reference/list/507.htm]|

===Evolution===
Gene pyramiding is considered one of the most effective strategies for achieving durable resistance against blast disease (Magnaporthe oryzae B. Couch) in rice (Oryza sativa L.), although few studies have evaluated the combining effect of the resistance genes.the development of pyramided lines with two major blast resistance genes, Pish and Pib, and the evaluation of the combining effect of them. The two genes pyramided lines were selected from the progenies of a cross between one near isogenic line (NIL), which harbours Pish, and another NIL, which harbours Pib, in the genetic background of blast susceptible variety, CO 39. The presence of the resistance genes was confirmed by DNA markers linked to them. To obtain DNA markers for Pish, we genetically mapped the Pish locus. We confirmed the additive effect of Pish and Pib in the pyramided lines by their reaction patterns to blast isolates, suggesting the potential availabilities of the combinations of these genes. In addition, we provide DNA markers linked to Pish for marker aided selection in rice blast resistance breeding.【1】Link = [http://www.ricedata.cn/reference/list/13405.htm]|

You can also add sub-section(s) at will.

Os05g0333200

2014-06-09T01:17:30Z

Zhangshoudong: /* References */

Please input one-sentence summary here.

==Annotated Information==
===Function===
The ''d1'' mutant, which is deficient for the heterotrimeric G-protein α subunit (G α ) gene of rice, shows dwarfi sm and sets small round seeds<ref name="ref1" />. Heterotrimeric G proteins, which are composed of α (G α ),β (G β ) and γ (G γ ) subunits, play a variety of roles in a wide range of physiological responses by transducing extracellular information to intracellular components<ref name="ref2" /><ref name="ref3" />. Heterotrimeric G proteins act as signal transducer between a receptor (G-protein-coupled receptors, GPCRs) and downstream effectors. G-protein signaling starts with a conformational change of the GPCR upon ligand perception.The GPCR is a guanine-nucleotide-exchange
factor (GEF) and its activation by the ligand promotes the exchange of GDP for GTP in the associated G α subunit. Subsequently, this complex dissociates into a G α -GTP monomer and a G β γ dimer to regulate downstream effectors. Mammals have multiples of each of the > 20 G α , 5 G β and 11 G γgenes<ref name="ref2" />.
[[File:Plant and cell morphology of d1-5.png|left|thumb|500px|''Plant and cell morphology of d1-5 (from reference <ref name="ref1" />).'']]

===Expression===
The G α protein was expressed in all the organs . Previously, the expression of the Arabidopsis G α(GPA1) was investigated by immunohistochemistry using anti-Arabidopsis G α antibody<ref name="ref4" /> and it was shown that G α is present throughout all development stages and in all organs examined such as roots, fl oral stems, rosette leaves, cauline leaves, fl owers and seed pods, with the exception of mature seeds. The accumulation of G α in Arabidopsis was higher in immature than in mature organs. The fact that the rice and Arabidopsis G α s were accumulated in the developing organs, fitted well the possibility that plant G α s are involved in the regulation of cell proliferation<ref name="ref1" />.
The expression pattern of rice G α suggests that this is also the case in rice. It was shown by immunochemical analysis that in Arabidopsis, G α accumulates highly in the root meristems, the shoot apical meristems and the floral meristems<ref name="ref4" />. The differences in expression in the meristem region between rice and Arabidopsis may refl ect speciesspecifi c expression patterns. The expression of the promoter of the Arabidopsis G α gene (GPA1) has also been investigated using histochemical analysis of transgenic plants expressing the GPA1::GUS reporter gene<ref name="ref5" />.

=== Evolution ===

Studies of proteins that interact with plant G α will also be important for understanding dwarfi sm in d1 . As previously mentioned, many proteins that interact with Arabidopsis G α were isolated, namely three GPCR-type proteins, GCR1,GTG1 and GTG2; one modulator, AtRGS1; four effectors, AtPrin1, AtPLD α 1, PD1 and THF1. It is not known whether rice homologues of these genes interact with rice G α or not<ref name="ref1" />.

== Labs working on this gene ==
1 Department of Bioscience, Fukui Prefectural University, 4-1-1 Matsuoka Kenjyojima, Eiheiji-cho, Yoshida-gun, Fukui, 910-1195 Japan

2 Bioscience and Biotechnology Center, Nagoya University, Chikusa, Nagoya, 464-8604 Japan

3 Department of Biology, University of North Carolina, Chapel Hill,North Carolina 27599, USA

4 Biology Department, Penn State University, University Park,Pennsylvania 16802-5301, USA

5 Cold Spring Harbor Laboratory, Cold Spring Harbor, New York 11724-2212

==References==
<references>
<ref name="ref1"> Izawa Y, Takayanagi Y, Inaba N, et al. Function and expression pattern of the α subunit of the heterotrimeric G protein in rice[J]. Plant and cell physiology, 2010, 51(2): 271-281.</ref>
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Function and Expression Pattern of the α Subunit of the Heterotrimeric G Protein in Rice
Plant and Cell Physiology, 2010, 51(2): 271-281
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A metastable DWARF1 epigenetic mutant affecting plant stature in rice
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Suppression of the heterotrimeric G protein causes abnormal morphology, including dwarfism, in rice
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Molecular Cloning and Characterization of a cDNA for the α Subunit of a G Protein from Rice
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