Os01g0194300 - Revision history

Xysj2019: /* References */

2017-03-06T06:30:02Z

‎References

Xysj2019: /* Evolution */

2017-03-06T06:29:40Z

‎Evolution

Xysj2019 at 06:28, 6 March 2017

2017-03-06T06:28:54Z

Xysj2012: /* Structured Information */

2015-05-13T06:59:11Z

‎Structured Information

Wangqiuqiu: /* Labs working on this gene */

2014-06-05T09:18:34Z

‎Labs working on this gene

Wangqiuqiu: /* References */

2014-06-05T09:15:44Z

‎References

Wangqiuqiu: /* Evolution */

2014-06-05T09:10:14Z

‎Evolution

Wangqiuqiu: /* Function */

2014-06-05T09:07:24Z

‎Function

Wangqiuqiu: /* Annotated Information */

2014-06-05T08:42:37Z

‎Annotated Information

Wangqiuqiu: /* Annotated Information */

2014-06-05T08:38:35Z

‎Annotated Information

← Older revision		Revision as of 06:30, 6 March 2017
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	</references>		</references>

		+	==Structured Information==

	[[Category:Genes]]		[[Category:Genes]]

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 ===Evolution===
-the role of OsNPR1 in plant defense response is similar to the role of AtNPR1but not the role ofNaNPR1. This finding is consistent with the result that At NPR1 is more homologous to OsNPR1 than is NaNPR1 in evolution and biochemistry. rice had a NPR1-mediated disease-resistance pathway similar to Arabidopsis.
+* The role of OsNPR1 in plant defense response is similar to the role of AtNPR1but not the role ofNaNPR1. This finding is consistent with the result that At NPR1 is more homologous to OsNPR1 than is NaNPR1 in evolution and biochemistry. rice had a NPR1-mediated disease-resistance pathway similar to Arabidopsis.
 ==Labs working on this gene==

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-Please input one-sentence summary here.
+The rice '''''Os01g0194300''''' was reported as '''''NH1''''' in 2005 <ref name="ref1" /> by researchers from U.S.A.
 ==Function==
-OsNPR1(also called NH1) may also play a central role in herbivoreinduced rice defense responses. It has been reported that transcript levels of OsNPR1are upregulated following infection by the bacterial blight Xanthomonas oryzae pv.oryzae (Xoo),the rice fungal blast Magnaporthe grisea and treatment with signal molecules, such as benzothiadiazole, methyl jasmonate and ET.OsNPR1 positively regulates expression of PR genes and rice resistance to bacterial blight disease  and fungal blast disease but negatively modulates resistance to the white-backed planthopper Sogatella furciferia.OsNPR1 may play a role in the reallocation of energy and resources during defense responses. However, how OsNPR1 mediates defenserelated signaling pathways and thus regulates the ability of rice to resist herbivores remains largely unknown.the rusult that OsNPR1 is involved in rice herbivore defense.
+* '''''NH1''''' may be involved in the regulation of SA in response to environmental changes.
 ==Expression==
-OsNPR1 in the herbivore-mediated induction of JA and ET pathways,and in the direct and indirect defenses of rice. transcript levels of OsNPR1 are upregulated when plants are treated by SA or JA.Interestingly, mechanical wounding enhances the mRNA levels of OsNPR1 quickly, whereas herbivore infestation elicits them more slowly ,much later than the time when the JA level in SSB-infested plants reaches its maximum (3 h after SSB infestation).It has been well documented that herbivore infestation elicits higher JA levels than does mechanical wounding in many plant species, including rice.Given an obvious inhibitory role ofOsNPR1on JA and ET biosynthesis ,it may be that rice plants perceive the herbivore infestation and then inhibit the increase in mRNA levels of OsNPR1at the early stage; thus,plants may prioritize JA- and ETdependent defense responses against herbivores. The increase in expression levels of OsNPR1at the later time points, which decreases the JA and ET levels, might be a regulation strategy of rice plants, resulting in appropriate defense responses according to the damage by the invader.
+* Transgenic rice plants overexpressing '''''OsNH1''''' (NH1ox) acquire high levels of resistance to Xanthomonas oryzae pv. oryzae. The resistance phe- notype is heritable and correlates with the presence of the transgene and reduced bacterial growth.
 ===Evolution===
 the role of OsNPR1 in plant defense response is similar to the role of AtNPR1but not the role ofNaNPR1. This finding is consistent with the result that At NPR1 is more homologous to OsNPR1 than is NaNPR1 in evolution and biochemistry. rice had a NPR1-mediated disease-resistance pathway similar to Arabidopsis.
 ==Labs working on this gene==
-State Key Laboratory of Rice Biology, Institute of Insect Sciences, Zhejiang University, Hangzhou
+* Department of Plant Pathology, University of California, Davis 95616, U.S.A.; 2 United States Department of Agriculture-Agricultural Research Service, Prosser, WA 99350, U.S.A.
 ==References==
-Fitzgerald HA, Chern MS, Navarre R, Ronald PC (2004)Overexpression of(At)NPR1in rice leads to a BTH- and environment-induced lesion mimic/cell death phenotype. Mol Plant Microbe Interact 17: 140–151
+<references>
-Yuan YX, Zhong SH, Li Q, Zhu ZR, Lou YG, Wang LY,Wang JJ, Wang MY, Li QL, Yang DL, He ZH (2007)Functional analysis of riceNPR1-like genes reveals that OsNPR1/NH1is the rice orthologue conferring disease resistance with enhanced herbivore susceptibility. PlantBiotechnol J 5: 313–324
+* <ref name="ref1">

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 Yuan YX, Zhong SH, Li Q, Zhu ZR, Lou YG, Wang LY,Wang JJ, Wang MY, Li QL, Yang DL, He ZH (2007)Functional analysis of riceNPR1-like genes reveals that OsNPR1/NH1is the rice orthologue conferring disease resistance with enhanced herbivore susceptibility. PlantBiotechnol J 5: 313–324
 [[Category:Genes]]
 [[Category:Japonica mRNA]]

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 ==Labs working on this gene==
-Please input related labs here.
+State Key Laboratory of Rice Biology, Institute of Insect Sciences, Zhejiang University, Hangzhou
 ==References==

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 ==References==
-Please input cited references here.
+Fitzgerald HA, Chern MS, Navarre R, Ronald PC (2004)Overexpression of(At)NPR1in rice leads to a BTH- and environment-induced lesion mimic/cell death phenotype. Mol Plant Microbe Interact 17: 140–151
 ==Structured Information==

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 ==Annotated Information==
 ===Evolution===
 Please input evolution information here.