Os09g0272900 - Revision history

Rice2012: /* Structured Information */

2015-06-12T09:38:45Z

‎Structured Information

Canghai91: /* Expression */

2014-06-11T01:57:58Z

‎Expression

Canghai91: /* Expression */

2014-06-11T01:57:41Z

‎Expression

Canghai91: /* Evolution */

2014-06-11T01:52:25Z

‎Evolution

Canghai91: /* Evolution */

2014-06-11T01:50:13Z

‎Evolution

Canghai91: /* Evolution */

2014-06-11T01:42:40Z

‎Evolution

Canghai91: /* Evolution */

2014-06-11T01:41:26Z

‎Evolution

Canghai91: /* Labs working on this gene */

2014-05-29T09:06:48Z

‎Labs working on this gene

Canghai91: /* Labs working on this gene */

2014-05-29T09:06:10Z

‎Labs working on this gene

Canghai91: /* References */

2014-05-29T09:02:28Z

‎References

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 ==Structured Information==
 [[Category:Genes]]
 [[Category:Japonica mRNA]]

← Older revision		Revision as of 01:57, 11 June 2014
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	The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection.		The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection.
		+
		+
		+
	[[File:Expression of PB.JPG]]		[[File:Expression of PB.JPG]]

← Older revision		Revision as of 01:57, 11 June 2014
Line 11:		Line 11:

	The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection.		The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection.
		+	[[File:Expression of PB.JPG]]

	===Evolution===		===Evolution===

← Older revision		Revision as of 01:52, 11 June 2014
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−	[[File::Evolution of PB.JPG]]	+	[[File:Evolution of PB.JPG]]

	==Labs working on this gene==		==Labs working on this gene==

← Older revision		Revision as of 01:50, 11 June 2014
Line 16:		Line 16:


−	[[File:Evolution of PB]]	+	[[File::Evolution of PB.JPG]]

	==Labs working on this gene==		==Labs working on this gene==

← Older revision		Revision as of 01:42, 11 June 2014
Line 16:		Line 16:


−	[[File:Evolution of PB]]	+	[[File:Evolution of PB]]

	==Labs working on this gene==		==Labs working on this gene==

@@ Line 13: / Line 13: @@
 ===Evolution===
+[[File:Evolution of PB]]
 ==Labs working on this gene==

@@ Line 21: / Line 21: @@
 [1]Shandong university of science and technology college of life science, shandong zibo 255049;
 [2]Shandong academy of agricultural sciences high-tech research center, shandong jinan 250100
 [3]Shandong academy of agricultural sciences high-tech research center, jinan, shandong province, 250100
 [4]Shandong academy of agricultural sciences high-tech research center, jinan, shandong province, 250100;
 [5]The ministry of agriculture of huanghuaihai laboratory of crop genetic improvement and biotechnology, shandong jinan 250100
 [6]Shandong university of science and technology college of life science, shandong zibo, 255049

@@ Line 18: / Line 18: @@
 ==Labs working on this gene==
-Please input related labs here.
 ==References==