Os11g0247300 - Revision history

Rice2012: /* References */

2015-06-13T09:05:17Z

‎References

2015-06-12T04:24:32Z

‎Structured Information

2014-06-06T04:57:31Z

‎Annotated Information

2014-06-06T04:49:25Z

2014-06-06T04:34:02Z

‎References

2014-06-06T04:32:33Z

‎Labs working on this gene

2014-06-06T04:31:04Z

‎Evolution

2014-06-06T04:30:15Z

‎Expression

2014-06-06T04:28:21Z

‎Function

2014-06-04T08:23:46Z

‎Function

← Older revision		Revision as of 09:05, 13 June 2015
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	[[Category:Japonica Chromosome 11]]		[[Category:Japonica Chromosome 11]]
	[[Category:Chromosome 11]]		[[Category:Chromosome 11]]
		+	==Structured Information==

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 Ashikari M, Matsuoka M (2006) Identification, isolation and pyramiding of quantitative trait loci for rice breeding. Trends Plant Sci 11:344-50
 [[Category:Genes]]
 [[Category:Japonica mRNA]]

← Older revision		Revision as of 04:57, 6 June 2014
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	==Annotated Information==		==Annotated Information==
		+	Small and round seed 5 gene encodes alpha-tubulin regulating seed cell elongation in rice
	===Function===		===Function===

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 Shuhei Segami1, Izumi Kono23, Tsuyu Ando24, Masahiro Yano5, Hidemi Kitano6, Kotaro Miura1* and Yukimoto Iwasaki1*
-CAS
+Faculty of Biotechnology, Fukui Prefectural University, 4-1-1 Kenjojima, Matsuoka, Eiheiji-Town, Fukui 910-1195, Japan
 ==References==

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 ==References==
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 ==Structured Information==

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 ==Labs working on this gene==
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 ==References==

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 ===Evolution===
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+In linkage analysis, we could not clearly distinguish seed size in F2 seed derived from distant cross between Srs5 and Kasalath. This was likely to be caused by background difference between indica and japonica. To overcome this, we used a chromosome segment substitution line (CSSL), which is a plant series that possesses relatively large chromosome segments of donor parent chromosomes in the recurrent parental chromosome background (Yano and Sasaki 1997, Yano 2001, Ebitani et al. 2005, Ashikari and Matsuoka 2006, Fukuoka et al. 2010). CSSLs can be used to achieve high accuracy in phenotyping in F2 populations. In fact, we could make classification of two seed size, wild and mutant type, in the F2 population derived from a cross between Srs5 and a CSSL.
-You can also add sub-section(s) at will.
+Genetic analysis of the F2 population derived from a cross between Srs5 and SL233 demonstrated that the Srs5 gene act as semi-dominant manner. This semi-dominant effect was also confirmed in complementation test. Although The Srs5 mutants carrying WT SRS5 gene showed longer seeds than that of the plants containing empty vector, the degree of recovery was not completely same as WT (Figure 4B). The reason that the rescue by WT SRS5 gene was partial may be due to compete between WT and mutation gene products or incomplete conformation of the tubulin complex.
 ==Labs working on this gene==

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 ===Expression===
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+Seed size and weight are important traits for rice yield (Song and Ashikari 2008, Takeda and Matsuoka 2008). Several quantitative trait loci (QTLs) affecting seed size have been identified, namely GW2 encoding a RING-type protein that functions as an E3 ubiquitin ligase (Song et al. 2007), qSW5 encoding a novel protein with no known domains (Shoumura et al. 2008), and GS3 encoding a membrane protein with various conserved domains (Fan et al. 2006, Takano-Kai et al. 2009). Loss of GW2 and qSW5 function leads to a wider seed phenotype, and loss of GS3 function leads to a longer seed phenotype, both resulting in increased yield.
 ===Evolution===

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 ==Annotated Information==
 ===Function===
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-MaP
+Seed size is an important trait in determinant of rice seed quality and yield. In this study, we report a novel semi-dominant mutant Small and round seed 5 (Srs5) that encodes alpha-tubulin protein. Lemma cell length was reduced in Srs5 compared with that of the wild-type. Mutants defective in the G-protein alpha subunit (d1-1) and brassinosteroid receptor, BRI1 (d61-2) also exhibited short seed phenotypes, the former due to impaired cell numbers and the latter due to impaired cell length. Seeds of the double mutant of Srs5 and d61-2 were smaller than those of Srs5 or d61-2. Furthermore, SRS5 and BRI1 genes were highly expressed in Srs5 and d61-2 mutants. These data indicate that SRS5 independently regulates cell elongation of the brassinosteroid signal transduction pathway.
 ===Expression===