Difference between revisions of "Os05g0121600"
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| − | + | The rice gene Os05g0121600 was reported as '''''RSR1''''' in 2010<ref name="ref1" /> by researchers from China. | |
==Annotated Information== | ==Annotated Information== | ||
| + | ===Gene Symbol=== | ||
| + | *'''''Os05g0121600 <=> OsRSR1,RSR1 | ||
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===Function=== | ===Function=== | ||
| − | + | * '''''Rice Starch Regulator1 (RSR1)''''', an APETALA2/ethylene-responsive element binding protein family transcription factor, was found to negatively regulate the expression of type I starch synthesis genes. | |
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| + | ===Mutation=== | ||
| + | [[File:Os05g0121600-1.png|right|thumb|400px|'''Figure 5. Endosperm starch contents and seed morphology of Zhonghua 11 (wild-type [WT]), rsr1 mutant, and RSR1-overexpressing (L1–L3) lines. A, AAC in endosperm.''' '' <ref name="ref1" />.'']] | ||
| + | * Seeds of the knockout mutant rsr1 consistently show the increased amylose content and altered fine structure of amylopectin and consequently form the round and loosely packed starch granules, resulting in decreased gelatinization temperature. In addition, rsr1 mutants have a larger seed size and increased seed mass and yield. | ||
| − | + | * RSR1 overexpression suppresses the expression of starch synthesis genes, resulting in altered amylopectin structure and increased gelatinization temperature. | |
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You can also add sub-section(s) at will. | You can also add sub-section(s) at will. | ||
| + | ==Labs working on this gene== | ||
| + | * National Key Laboratory of Plant Molecular Genetics, Institute of Plant Physiology and Ecology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200032, China | ||
| + | ==References== | ||
| + | <references> | ||
| + | * <ref name="ref1"> | ||
| + | Fu FF, Xue HW. Coexpression analysis identifies Rice Starch Regulator1, a rice | ||
| + | AP2/EREBP family transcription factor, as a novel rice starch biosynthesis | ||
| + | regulator. Plant Physiol. 2010 Oct;154(2):927-38. doi: 10.1104/pp.110.159517. | ||
| + | Epub 2010 Aug 16. PubMed PMID: 20713616; PubMed Central PMCID: PMC2949045. | ||
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| + | </ref> | ||
| + | </references> | ||
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==Structured Information== | ==Structured Information== | ||
| − | + | [[Category:Genes]][[Category:Oryza Sativa Japonica Group]][[Category:Japonica Chromosome 5]] | |
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| − | [[Category:Genes]] | ||
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| − | [[Category:Oryza Sativa Japonica Group]] | ||
| − | [[Category:Japonica | ||
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Latest revision as of 07:08, 16 October 2016
The rice gene Os05g0121600 was reported as RSR1 in 2010[1] by researchers from China.
Contents
Annotated Information
Gene Symbol
- Os05g0121600 <=> OsRSR1,RSR1
Function
- Rice Starch Regulator1 (RSR1), an APETALA2/ethylene-responsive element binding protein family transcription factor, was found to negatively regulate the expression of type I starch synthesis genes.
Mutation
Figure 5. Endosperm starch contents and seed morphology of Zhonghua 11 (wild-type [WT]), rsr1 mutant, and RSR1-overexpressing (L1–L3) lines. A, AAC in endosperm. [1].
- Seeds of the knockout mutant rsr1 consistently show the increased amylose content and altered fine structure of amylopectin and consequently form the round and loosely packed starch granules, resulting in decreased gelatinization temperature. In addition, rsr1 mutants have a larger seed size and increased seed mass and yield.
- RSR1 overexpression suppresses the expression of starch synthesis genes, resulting in altered amylopectin structure and increased gelatinization temperature.
You can also add sub-section(s) at will.
Labs working on this gene
- National Key Laboratory of Plant Molecular Genetics, Institute of Plant Physiology and Ecology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200032, China
References
- ↑ 1.0 1.1 Fu FF, Xue HW. Coexpression analysis identifies Rice Starch Regulator1, a rice AP2/EREBP family transcription factor, as a novel rice starch biosynthesis regulator. Plant Physiol. 2010 Oct;154(2):927-38. doi: 10.1104/pp.110.159517. Epub 2010 Aug 16. PubMed PMID: 20713616; PubMed Central PMCID: PMC2949045.