Difference between revisions of "Os11g0684000"
(Created page with "{{JaponicaGene|
GeneName = Os11g0684000|
Description = Similar to Transcription factor MYB21 (Myb-related protein 21) (AtMYB21) (Myb homolog 3) (AtMyb3)|
Version = NM_00107...") |
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| − | + | The rice '''Os11g0684000''' was reported as ''' OsMPK5''' in 2010 <ref name="ref1" /> by researchers from the USA. | |
| − | + | <br> | |
| − | + | ==Annotated Information== | |
| − | + | ===Gene Symbol=== | |
| − | + | *'''Os11g0684000 <=> OsMPK5''' | |
| − | + | <br> | |
| − | + | ===Function=== | |
| + | *These data favor a model whereby exogenous ABA enhances resistance against C. miyabeanus at least in part by suppressing pathogen-induced ET action in an '''OsMPK5'''-dependent manner. | ||
| + | <br> | ||
| + | ===Evolution=== | ||
| + | *Exogenous ethephon application enhances susceptibility, whereas genetic disruption of ET signaling renders plants less vulnerable to C. miyabeanus attack, thereby inducing a level of resistance similar to that observed on ABA-treated wild-type plants. Moreover, ABA treatment alleviates C. miyabeanus-induced activation of the ET reporter gene EBP89, while derepression of pathogen-triggered EBP89 transcription via RNA interference-mediated knockdown of '''OsMPK5''',an ABA-primed mitogen-activated protein kinase gene, compromises ABA-IR. | ||
| + | <br> | ||
| − | + | ==Labs working on this gene== | |
| − | + | *Laboratory of Phytopathology, Faculty of Bioscience Engineering, Ghent University, B–9000 Ghent, Belgium (D.D.V., M.H.); | |
| − | + | *Department of Plant Pathology and Huck Institutes of Life Sciences, Pennsylvania State University, University Park, Pennsylvania 16802 (Y.Y.); | |
| − | + | *Plant Breeding, Genetics, and Biotechnology Division, International Rice Research Institute, 1099 Manila, Philippines (C.V.C.) | |
| − | + | <br> | |
| − | + | ==References== | |
| − | + | <references> | |
| − | + | <ref name="ref1"> | |
| − | + | Abscisic Acid-Induced Resistance against the Brown Spot Pathogen Cochliobolus miyabeanus in Rice Involves MAP Kinase-Mediated Repression of Ethylene Signaling | |
| − | + | </ref> | |
| − | + | ||
| − | + | </references> | |
| − | < | + | <br> |
| − | + | ==Structured Information== | |
| − | name= | + | [[Category:Genes]][[Category:Oryza Sativa Japonica Group]][[Category:Japonica Chromosome 11]] |
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| − | </ | ||
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| − | [[Category:Genes]] | ||
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| − | [[Category:Oryza Sativa Japonica Group]] | ||
| − | [[Category:Japonica | ||
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Latest revision as of 05:51, 12 December 2016
The rice Os11g0684000 was reported as OsMPK5 in 2010 [1] by researchers from the USA.
Contents
Annotated Information
Gene Symbol
- Os11g0684000 <=> OsMPK5
Function
- These data favor a model whereby exogenous ABA enhances resistance against C. miyabeanus at least in part by suppressing pathogen-induced ET action in an OsMPK5-dependent manner.
Evolution
- Exogenous ethephon application enhances susceptibility, whereas genetic disruption of ET signaling renders plants less vulnerable to C. miyabeanus attack, thereby inducing a level of resistance similar to that observed on ABA-treated wild-type plants. Moreover, ABA treatment alleviates C. miyabeanus-induced activation of the ET reporter gene EBP89, while derepression of pathogen-triggered EBP89 transcription via RNA interference-mediated knockdown of OsMPK5,an ABA-primed mitogen-activated protein kinase gene, compromises ABA-IR.
Labs working on this gene
- Laboratory of Phytopathology, Faculty of Bioscience Engineering, Ghent University, B–9000 Ghent, Belgium (D.D.V., M.H.);
- Department of Plant Pathology and Huck Institutes of Life Sciences, Pennsylvania State University, University Park, Pennsylvania 16802 (Y.Y.);
- Plant Breeding, Genetics, and Biotechnology Division, International Rice Research Institute, 1099 Manila, Philippines (C.V.C.)
References
- ↑ Abscisic Acid-Induced Resistance against the Brown Spot Pathogen Cochliobolus miyabeanus in Rice Involves MAP Kinase-Mediated Repression of Ethylene Signaling