Difference between revisions of "Os04g0477300"

From RiceWiki
Jump to: navigation, search
 
(3 intermediate revisions by the same user not shown)
Line 1: Line 1:
−
Please input one-sentence summary here.
+
The rice gene Os04g0477300 was reported as '''''BET1''''' in 2011<ref name="ref1" /> by researchers from Japan.
  
 
==Annotated Information==
 
==Annotated Information==
 +
===Gene Symbol===
 +
*'''''Os04g0477300 <=> BET1, ONAC081, ONAC81, NAC81
 +
 
===Function===
 
===Function===
−
Please input function information here.
+
* '''''BORON EXCESS TOLERANT1''''' is a gene responsible for tolerance to boron (B) toxicity in rice (Oryza sativa).
 +
* '''''BET1''''' gene encodes a NAC (NAM, ATAF, and CUC)-like transcription factor and the function of the transcript is abolished in B-toxicity-tolerant cultivars.
  
−
===Expression===
+
===Expression ===
−
Please input expression information here.
+
* Quantitative RT-PCR analysis revealed that '''''BET1''''' gene was expressed mainly in the roots and the expression level was less in the shoots in 7-d-old seedlings.
  
 
===Evolution===
 
===Evolution===
−
Please input evolution information here.
 
  
 
You can also add sub-section(s) at will.
 
You can also add sub-section(s) at will.
 +
==Labs working on this gene==
 +
* Laboratory of Plant Nutrition, Division of Applied Life Sciences (K.O., T.M.) and Laboratory of Breeding, Division of Applied Biologies (Y.O.), Graduate School of Agriculture, Kyoto University, Kyoto 606–8502, Japan;
 +
* Department of Biological Resources Management, School of Environmental Science, University of Shiga Prefecture, Hikone 522–8533, Japan (A.S);
 +
* Laboratory of Plant Nutrition and Fertilizers, Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, University of Tokyo, Tokyo 113–8657, Japan (T.F.)
 +
==References==
 +
<references>
 +
* <ref name="ref1">
 +
Ochiai K, Shimizu A, Okumoto Y, Fujiwara T, Matoh T. Suppression of a NAC-like
 +
transcription factor gene improves boron-toxicity tolerance in rice. Plant
 +
Physiol. 2011 Jul;156(3):1457-63. doi: 10.1104/pp.110.171470. PubMed PMID:
 +
21543724; PubMed Central PMCID: PMC3135931.
  
−
==Labs working on this gene==
+
</ref>
−
Please input related labs here.
+
</references>
  
−
==References==
 
−
Please input cited references here.
 
  
 
==Structured Information==
 
==Structured Information==
 
     [[Category:Genes]][[Category:Oryza Sativa Japonica Group]][[Category:Japonica Chromosome 4]]
 
     [[Category:Genes]][[Category:Oryza Sativa Japonica Group]][[Category:Japonica Chromosome 4]]

Latest revision as of 01:59, 13 December 2016

The rice gene Os04g0477300 was reported as BET1 in 2011[1] by researchers from Japan.

Annotated Information

Gene Symbol

  • Os04g0477300 <=> BET1, ONAC081, ONAC81, NAC81

Function

  • BORON EXCESS TOLERANT1 is a gene responsible for tolerance to boron (B) toxicity in rice (Oryza sativa).
  • BET1 gene encodes a NAC (NAM, ATAF, and CUC)-like transcription factor and the function of the transcript is abolished in B-toxicity-tolerant cultivars.

Expression

  •  Quantitative RT-PCR analysis revealed that BET1 gene was expressed mainly in the roots and the expression level was less in the shoots in 7-d-old seedlings.

Evolution

You can also add sub-section(s) at will.

Labs working on this gene

  • Laboratory of Plant Nutrition, Division of Applied Life Sciences (K.O., T.M.) and Laboratory of Breeding, Division of Applied Biologies (Y.O.), Graduate School of Agriculture, Kyoto University, Kyoto 606–8502, Japan;
  • Department of Biological Resources Management, School of Environmental Science, University of Shiga Prefecture, Hikone 522–8533, Japan (A.S);
  • Laboratory of Plant Nutrition and Fertilizers, Department of Applied Biological Chemistry, Graduate School of Agricultural and Life Sciences, University of Tokyo, Tokyo 113–8657, Japan (T.F.)

References

  1. ↑ Ochiai K, Shimizu A, Okumoto Y, Fujiwara T, Matoh T. Suppression of a NAC-like transcription factor gene improves boron-toxicity tolerance in rice. Plant Physiol. 2011 Jul;156(3):1457-63. doi: 10.1104/pp.110.171470. PubMed PMID: 21543724; PubMed Central PMCID: PMC3135931.


Structured Information