Difference between revisions of "Os02g0731700"
(Created page with "{{JaponicaGene|
GeneName = Os02g0731700|
Description = Similar to CONSTANS-like 1 protein|
Version = NM_001054557.1 GI:115448484 GeneID:4330626|
Length = 1948 bp|
Definit...") |
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| − | + | The rice Os02g0731700 was reported as '''''Ghd2''''' in 2016 by researchers from china. <ref name="ref1" /> | |
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| − | + | ==Annotated Information== | |
| − | + | *'''''Os02g0731700''''' '''''<=>''''' '''''Ghd2, OsBBX8, OsCCT09, OsK, OsK/Ghd2''''' | |
| − | + | ===Function=== | |
| − | + | *Ghd2 functions as a regulator by integrating environmental signals with the senescence process into a developmental programme through interaction with different proteins | |
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| − | + | ===Expression=== | |
| − | + | *Ghd2 is expressed mainly in the rice (Oryza sativa) leaf with the highest level detected at the grain-filling stage, and it is down-regulated by drought stress conditions. | |
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| − | + | ===Phenotypic analysis=== | |
| − | + | *Overexpression of Ghd2 resulted in significantly reduced drought resistance, while its knockout mutant showed the opposite phenotype. | |
| − | + | *The earlier senescence symptoms and the transcript up-regulation of many senescence-associated genes (SAGs) in Ghd2-overexpressing transgenicrice plants under drought stress conditions indicate that Ghd2 plays essential roles in accelerating drought-induced leaf senescence in rice. | |
| − | + | *developmental and dark-induced leaf senescence was accelerated in the Ghd2-overexpressing rice and delayed in the ghd2 mutant. | |
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| − | + | ==Labs working on this gene== | |
| − | + | *National Key Laboratory of Crop Genetic Improvement and National Center of Plant Gene Research (Wuhan), Huazhong Agricultural | |
| − | + | University, Wuhan 430070, China | |
| − | < | + | |
| − | + | ==References== | |
| − | + | <references> | |
| − | + | <ref name="ref1"> | |
| − | + | Liu J, Shen J, Xu Y, Li X, Xiao J, Xiong L. Ghd2, a CONSTANS-like gene, | |
| − | + | confers drought sensitivity through regulation of senescence in rice. J Exp Bot. | |
| − | [[Category:Genes]] | + | 2016 Oct;67(19):5785-5798. Epub 2016 Sep 16. PubMed PMID: 27638689; PubMed |
| − | + | Central PMCID: PMC5066496. | |
| − | [[Category:Oryza Sativa Japonica Group]] | + | </ref> |
| − | [[Category:Japonica | + | </references> |
| − | + | ||
| − | + | ==Structured Information== | |
| + | [[Category:Genes]][[Category:Oryza Sativa Japonica Group]][[Category:Japonica Chromosome 2]] | ||
Latest revision as of 03:49, 20 May 2017
The rice Os02g0731700 was reported as Ghd2 in 2016 by researchers from china. [1]
Contents
Annotated Information
- Os02g0731700 <=> Ghd2, OsBBX8, OsCCT09, OsK, OsK/Ghd2
Function
- Ghd2 functions as a regulator by integrating environmental signals with the senescence process into a developmental programme through interaction with different proteins
Expression
- Ghd2 is expressed mainly in the rice (Oryza sativa) leaf with the highest level detected at the grain-filling stage, and it is down-regulated by drought stress conditions.
Phenotypic analysis
- Overexpression of Ghd2 resulted in significantly reduced drought resistance, while its knockout mutant showed the opposite phenotype.
- The earlier senescence symptoms and the transcript up-regulation of many senescence-associated genes (SAGs) in Ghd2-overexpressing transgenicrice plants under drought stress conditions indicate that Ghd2 plays essential roles in accelerating drought-induced leaf senescence in rice.
- developmental and dark-induced leaf senescence was accelerated in the Ghd2-overexpressing rice and delayed in the ghd2 mutant.
Labs working on this gene
- National Key Laboratory of Crop Genetic Improvement and National Center of Plant Gene Research (Wuhan), Huazhong Agricultural
University, Wuhan 430070, China
References
- ↑ Liu J, Shen J, Xu Y, Li X, Xiao J, Xiong L. Ghd2, a CONSTANS-like gene, confers drought sensitivity through regulation of senescence in rice. J Exp Bot. 2016 Oct;67(19):5785-5798. Epub 2016 Sep 16. PubMed PMID: 27638689; PubMed Central PMCID: PMC5066496.
