Difference between revisions of "Os09g0272900"
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===Expression=== | ===Expression=== | ||
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| + | The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection. | ||
===Evolution=== | ===Evolution=== | ||
Revision as of 08:57, 29 May 2014
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Contents
Annotated Information
Function
A prerequisite for the plant to induce defence response is the recognition of microbes and putative pathogens. Therefore, plants detect conserved microbial molecules called pathogen-(or microbe-) associated molecular patterns (PAMPs/MAMPs) as well as damage-associated molecular patterns.DAMPs are endogenous elicitors of the plant and are produced in consequence of the damage that is caused by plant-colonizing pathogens. However, resistance
as a consequence of PAMP-induced defence responses is designated as PAMP-triggered immunity (PTI). PAMPs are usually epitopic structures within molecules and are essential structural components for the microbe. They are present in many microbial species, and are, while not existing in the potential host, perceived by a broad spectrum of host species. Some typical bacterial PAMPs are lipopolysaccharides (LPS), peptidoglycans (PGNs), flagellin, and the elongation factor Tu (EF-Tu). LPS are found in the outer membrane of Gram-negative bacteria, and plant cells predominantly recognise the conserved lipid A but also the core oligosaccharide and the O-antigen structure of LPS.
Expression
The expression of PR genes is often a result of triggered SA-, JA- or ethylene-dependent defence pathways. PR-proteins are classified in at least 17different families based on their primary structure.Many PR protein families possess direct antimicrobial activities: proteins of the PR3, PR4 and PR-11 families work as chitinases, whereas the PR-2 family represent β-1,3-glucanases. By contrast, the microbial targets of many PR-families are still unkown. How is PR gene expression activated? PR-1 gene expression is predominantly triggered by the SA-pathway and, exogenous application of SA is sufficient to induce PR-1 expression. NPR-1 (NON-EXPRESSOR OF PR GENES1) is acentral mediator of SA signalling and PR gene expression. The corresponding mutant impaired in NPR-1 expression does not respond to SA application by PR gene expression, but accumulates SA after pathogen infection.
Evolution
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Labs working on this gene
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References
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Structured Information
| Gene Name |
Os09g0272900 |
|---|---|
| Description |
Similar to Skp1 (Fragment) |
| Version |
NM_001069277.1 GI:115478293 GeneID:4346585 |
| Length |
19453 bp |
| Definition |
Oryza sativa Japonica Group Os09g0272900, complete gene. |
| Source |
Oryza sativa Japonica Group ORGANISM Oryza sativa Japonica Group
Eukaryota; Viridiplantae; Streptophyta; Embryophyta; Tracheophyta;
Spermatophyta; Magnoliophyta; Liliopsida; Poales; Poaceae; BEP
clade; Ehrhartoideae; Oryzeae; Oryza.
|
| Chromosome | |
| Location |
Chromosome 9:6037570..6057022 |
| Sequence Coding Region |
6037764..6038320,6046484..6046523 |
| Expression | |
| Genome Context |
<gbrowseImage1> name=NC_008402:6037570..6057022 source=RiceChromosome09 preset=GeneLocation </gbrowseImage1> |
| Gene Structure |
<gbrowseImage2> name=NC_008402:6037570..6057022 source=RiceChromosome09 preset=GeneLocation </gbrowseImage2> |
| Coding Sequence |
<cdnaseq>atgattacgctgagcagtcctgtgatgtgtttcatccatgcgtcgtccgtgttcgaccgatcaatggcggcacccgcagcaccgcaggagaccaacgccgcggccggtgagatggtcacgctgatcagctcggacggagctcgcttcgaggtgccggaggcggcggtgaggctgtcgcagacggtgctcgacgagatgaagaaggacgactacaacgccatcaacggcatccctctccccaacgtcgccggcgacgtcctcgccaaggtggtcgagtactgcaccaagcacgcctccgccgccgccgccgccatcaacgccgacacggcgaagacgagcaaggaggaggaggagctgatgatgaagagcttcgacgacgagttcatcctggtggacaaccacatgctgtacagcttgctcacggcggcggacgccatgcgcatccaggggctgatggacctggcatgccagcgattggcggacatgatcaagggcaagacgtcggagcagatgaggcagacgttggggatcaccaacgacttcacgccggaggaagaagaggagtttcgccgggaggacgaggaacaatggctctga</cdnaseq> |
| Protein Sequence |
<aaseq>MITLSSPVMCFIHASSVFDRSMAAPAAPQETNAAAGEMVTLISS DGARFEVPEAAVRLSQTVLDEMKKDDYNAINGIPLPNVAGDVLAKVVEYCTKHASAAA AAINADTAKTSKEEEELMMKSFDDEFILVDNHMLYSLLTAADAMRIQGLMDLACQRLA DMIKGKTSEQMRQTLGITNDFTPEEEEEFRREDEEQWL</aaseq> |
| Gene Sequence |
<dnaseqindica>18703..19259#10500..10539#attcggctctcgctccttttcttggcgcttatatttttcttttcacatcaaactagcttctcaagctgtttagctacatagaattttttttcctctgtactctcggattactgattttcagcttggacaagtactgcttttcttgatcttttgctttacatggaggaattatagtttttctccatactatccctaataatcatatctcatcttccaataggagggtattactattgattctgcatctctcttctacccgctgcaacaatttgaagctagtaaactgttcagtttacaggtccccttgactgttacaggtatgcatcgtttcatcaaatttgtacatatgattttcttatttacctcaagcctcaaggtgtgttatacaacacaattcctagattttctatatgcatatgtccacagaaaagataatactggtcatatgtttgttttttacgtctggttaaacaaacaatcctagaaaaactattattataggcggtcaataattcatttgcacaagtttagctaaccagcatgtagaaattaaataatttgtattggaggattccacaacggtatgtggatatcaatttccagttctattaagaacccacccacggaaatgaattttcaactttttgttaatgtaggcgaaagaaaaacacattttaccaggcaactatacactgtcactggtcgcaaatacgaagtcacagtgatttcttacacgaaatatgcattttatcaaaagtcacaggtcgcaaatcagaagtcactaatacctatttcatattacaaatattttaaaatggaattatcaccaataacaaagttgttgatctcggccagctctttagtttttaatatagggcatatctcccgctgaggctgtttgaaaactttttttaaaaaaacatttttagagttgtttctatttctagacacggtttgttaggttagccgtcagcactaatcatttattccgttagtttttcagaggttctcccgtggaaacaagttgctttaaaatattaaataaatctatttttcaagtttcatataattaaaacttacttaatccagtgctaatgattttctcgtttttgttcgcacttctctccggatcaaacgtagctaatagtgtagacagttagttacagggtctgcctgtcaaaaccgttactcattatttgaaaatgccaaaactttgaaaattgatagaaatttgattttaagtccagtattagtgaatcaaagttctagctctcaacaagatctatctctttgagactgtatagagtccaaatattcgttataagctcttagatctatatttggaaacaaatttatgaattttttgaatgcccttaaatggagatgtgttctgtaatatgtattgaatttgtaaagctcgatgagattctacaactttataactttactggtgataattttttatttgatgtcatttatcctcccaaatatgctaaatgacatgaggtaaggttatgagaggtaatcctctttaccgcccatgtgcatgcttcacgtgaaaaattgcgtgactcgttgttttgtgaaatgtgaattattttattggtttttttttcatacaacatttaaaaactagaaatacatttttctcacgtgactggaatttatctgtacaaaatcgttgatttctacttgtcttagtcataggcggttggctaaacgcctgtaggaataatgggttaccgtcttaccgacctcctatacaatttttttatgtaccaatcattatttcgactattcttcctgaactggagctaggttatactaaaatgataatttatttaacagaaaatagtagtcactataaattgatatgttccaagttgataaaactacattcatttttgttttttattaaattatactccttgtatcccaaataaggtttaaccactttttacttatgtctcatcatataagacaggcaggcatgcatacattaattatcacctcttagttctttaaatttggtttactttaaattctccaccattaaacccccaatcacattgtttgcatgtatggatgtgatccaatcaaggaggtgattaaattctttcttgttctttgtggtagtagtggttgagccttatacttttagatggagagaataaatatatactataggcaccattagtttttttttgtctgtactatgttccacatttttctgtaactctatggaccgaatcgtcttcacgcttgagtcgatatgaacccggccgagtaacttcagagatgatgaaaggtccctcccataagggagataacttatgtcgaccccgtgtagtttaaatcttcctcaagaccatatctccgactagaaatgcacgtgatatgacattgcgattatgataacgactgaattaatgttgcctcacgagcttcttccaattggttgagatcatccactcggtcttcctcgtagcgttcctctttaacattgcgaaatctcatggactcaaactccacttcgcttggcaacattgcttctactccgtaaactaagaaaaacggcgactagcccgtagcccgactgggcgtagtgcgcaaagaccaaagtactgacggcagctgctcaacccatttgccagcatagggacgcagtcggttaaaaaaccgtgccttgatgccttgaagtaccacaccattggcgtgttcaacttgaccattgctcataggatgcgctacagaagcataacatatcttgatactaaagtcttcacaaaaatctttaaaaactccaccagtgaattgggtaccattatctgtaatgattcgattcggcaccccaaaccgatgcacaatgttgataaagaagtctcgagctttgttggctgtaatagttacaactggtttggcttcaatccatttggagaacttatcaacagccacaaaaagttgattaaatcctccggtagcccttttgaaaggaccaaccatgtcaagcccccaaaccgcaaatggccaggaaagagggatggtttgcaactcttggtccggcaaatgaatttgcctagcaaaaaattgacaactttcacacgttcgcacaaccttctcggcatcagacaccgctgtaggccagaaaaaaccctgtcgatatgctttgccgactatcgttcgtgctgccgcatgatttccacaaatgacagaatgaatatcttacaacaattgcctaccttcttctagagaaatatagcaatgtaggattccagatggactctttttgtataaatcggcctcaatcatgacataaagcttgttgtgtcgagaaatccgctcggcttcattcttgtcttgaggcaactcttgctgagttagaaatttgatgattggctctctccagtcggcatcaattactcctacttcattagcctctccatctccgactttatcctttcttgatactgtcagctcatagagatgctcgacaaaaatatcagaaggagctgcttctcgttttgacccaaaattagctaatctatcggctgcttcattgttgtgtctaagcacatgagttaactctcgaccatcaaatttatcttccaatttgcggacttcttgtcgatatgcaaccatattgtcgtcgagacaggaccaatctttcatgacttgattgacaaccaactatgaatcaccacgaaccactagatgtcagatccctagggatacaacaatcttcagcacatggagtaacgtctcatactctgccacattgtgggatgcagaaaagtgtatccacagcacgtagcttaacctttctccagtcggaaaaatcaaaacgacccctgctccggtgcccgatagtctctttgacccgtcaaaatgcatagtccaatactccaatttctccacaggtgtgtcttcttgacactcggtccactcggcaacaaaatctgctagtgcttgagatttgattgaagttcgcagcttgaaagataaatccaaagacatcaactccaaggcccacttcgcaatacgtccagttgcctctcgattatgaagtatgtcaccaagtggaaaagatgtgagcaccgtgatcgagtgactctgaaaataatgggataatttcctgacggtaattaagacaccatataataatttatgtacctggggatatcttattttggagtcggccaagacctcgctaacgaagtaaattggtcgttgaactttttgaacatggccgtcctcttctcgcttgacaaccaaaaccgtgcttaccacttgcgatgttgctgacacataaaacaacaatggctcttgcgggtgtggtgacgctaggattggtggagttgagagaaatcttttgaaatcattgaaggcttgctgagcttcgggtccccattgtaaattgtcagtcttcttgagcagtttgaagaagggcatccctcgctcgtcaagtcttgaaataaaccgactgagcgccgccatgcatccagttagcttctgaacgtctttctaggaactaggcggtttcatgttgaggatggcattgattttctccgggttggcttgtatgcccctatgggacatcataaagccaagcagcttccctgacggtactccgaaggtacacttttcagggttcaacttcatcctgaaagcacggatgctagcaaaggtttcctccaaatcagcaattaagtcatccttctgcttggtcttgatgactacatcatcaactacatcatctgagttgaaaaacatctctggattatgtgttgataagttgctctagcattcttcaatcaaaacggcatggtaatgtagcagtaagctccaaatggtgtgatgaatgaggtcttcgagcagttggattccttcaacctgatctggcggtatcccgagtagcaatccaaaaatctgagtaactcgcagccggttgttgagtcaacta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