Natural killer cell cytotoxicity and the multidrug resistance gene.

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P N Markham, J S Coon, A S Chong, H M Gebel

Author Information

P N Markham: Department of Immunology, Rush-Presbyterian-St Luke's Medical Center, Chicago, Illinois 60612.

PMID: 7679846

No abstract text available.

CD8 Antigens

Chloroquine

Cyclosporine

Cytotoxicity, Immunologic

DNA

Drug Resistance

Humans

Killer Cells, Natural

Membrane Glycoproteins

Perforin

Polyethylene Glycols

Polymerase Chain Reaction

Pore Forming Cytotoxic Proteins

RNA

Stearic Acids

T-Lymphocytes, Cytotoxic

Verapamil

CD8 Antigens

Membrane Glycoproteins

Pore Forming Cytotoxic Proteins

Stearic Acids

Perforin

Polyethylene Glycols

Solutol HS 15

RNA

Cyclosporine

Chloroquine

DNA

Verapamil

Journal Article

Resistance of multidrug-resistant lines to natural killer-like cell-mediated cytotoxicity.Resistance against natural killer cell cytotoxicity: analysis of mechanisms.Natural killer cell cytotoxicity: role of calmodulin.Mechanism of natural killer cell-mediated cytotoxicity.Thermal stresses reduce natural killer cell cytotoxicity.Human neonatal natural killer cell cytotoxicity function.Cystatin F Affects Natural Killer Cell Cytotoxicity.Antibody-dependent cell-mediated cytotoxicity and natural killer-cell phenomenon.[Inhibition of natural killer and interleukin 2-activated natural killer cell cytotoxicity by monosaccharides and lectins].Adriamycin-induced resistance to natural killer (NK)-mediated cytotoxicity.

Activation of human peripheral blood T cells does not lead to increased P-glycoprotein expression.Inhibition of cell-mediated cytolysis and P-glycoprotein function in natural killer cells by verapamil isomers and cyclosporine A analogs.

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