Alprazolam dependence prevented by substituting with the beta-carboline abecarnil.

G Pinna, R Galici, H H Schneider, D N Stephens, L Turski
Author Information
  1. G Pinna: Research Laboratories of Schering AG, Berlin, Germany.

Abstract

Abrupt termination of the treatment of humans with benzodiazepines (BDZs) leads to a rapid onset of discontinuation syndrome characterized by anxiety, muscle spasms, and occasionally convulsions. For this reason, it is recommended in clinical practice to reduce the dose of the BDZs gradually at the end of treatment. Nevertheless, many clinicians report signs of dependence even during gradual reduction of doses (tapering) of the BDZs in a large proportion of patients. Thus, there is considerable interest in discovering means of weaning patients away from BDZs without the risk of discontinuation syndrome. In the present study, mice withdrawn from chronic treatment with alprazolam showed anxiety, muscle rigidity, and seizures between days 1 and 28 after termination of the treatment. Replacement of alprazolam with the beta-carboline abecarnil for 7 days prevented the occurrence of the signs of dependence. In contrast, substitution of the beta-carboline antagonist ethyl-5-isopropoxy-4-methyl-beta-carboline-3-carboxylate (ZK93426) for alprazolam worsened the discontinuation syndrome. Replacement therapy with abecarnil after long-term treatment with the BDZs offers a novel method for rapid tapering.

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MeSH Term

Alprazolam
Analysis of Variance
Animals
Anticonvulsants
Anxiety
Carbolines
Electroencephalography
Electromyography
Male
Mice
Mice, Inbred Strains
Substance Withdrawal Syndrome
Substance-Related Disorders
Time Factors

Chemicals

Anticonvulsants
Carbolines
ZK 93426
abecarnil
Alprazolam

Word Cloud

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