Fractalkine, a CX3C chemokine, as a mediator of ocular angiogenesis.

Jian-Jang You, Chang-Hao Yang, Jen-Shang Huang, Muh-Shy Chen, Chung-May Yang
Author Information
  1. Jian-Jang You: Department of Ophthalmology, Keelung General Hospital, Department of Health, The Executive Yuan, Keelung, Taiwan.

Abstract

PURPOSE: Fractalkine (FKN) is a chemoattractant and adhesion molecule for leukocytes. Angiogenic effect of FKN also has been reported. This study was an investigation of FKN-mediated angiogenesis in vitro and in vivo to determine its role in ocular angiogenic disorders.
METHODS: FKN effects on cultured human umbilical vein endothelial cells (HUVECs) and bovine retinal capillary endothelial cells (BRECs) were evaluated with chemotaxis assay and a synthetic matrix capillary tube formation assay in vitro. Reverse transcription-polymerase chain reaction (RT-PCR) and Western blot analysis were used to detect mRNA and protein expression of FKN and its receptor, CX3CR1, in HUVECs and BRECs. A rabbit corneal neovascularization assay and an oxygen-induced retinopathy (OIR) model of mice were used to test the angiogenic property of FKN in vivo. FKN levels of vitreous samples from patients with proliferative diabetic retinopathy were measured by enzyme-linked immunosorbent assay (ELISA). Immunodepletion of FKN in PDR vitreous samples by anti-FKN polyclonal antibody was observed in endothelial cell chemotaxis assays.
RESULTS: FKN significantly induced migration of HUVECs and BRECs. FKN induced formation of endothelial cell capillary tubes on synthetic matrix. Expression of FKN and CX3CR1 was detected in HUVECs and BRECs by RT-PCR and Western blot analysis. FKN significantly induced more blood vessel growth than did the control in the rabbit corneal pocket neovascularization assay. Intravitreal injection of anti-mouse FKN antibody decreased retinal angiogenesis in the OIR model. The vitreous level of FKN was elevated in patients with PDR compared with control subjects. Immunodepletion of soluble FKN from PDR vitreous samples caused 36.6% less migration of BRECs.
CONCLUSIONS: FKN is an angiogenic mediator in vitro and in vivo. The vitreous level of FKN was elevated in patients with PDR. FKN may play an important role in ocular angiogenic disorders such as PDR.

MeSH Term

Angiogenesis Inducing Agents
Animals
Animals, Newborn
Blotting, Western
CX3C Chemokine Receptor 1
Cattle
Cells, Cultured
Chemokine CX3CL1
Chemotaxis
Corneal Neovascularization
Diabetic Retinopathy
Disease Models, Animal
Endothelium, Vascular
Enzyme-Linked Immunosorbent Assay
Female
Humans
Male
Mice
Mice, Inbred C57BL
Neovascularization, Physiologic
Oxygen
Pregnancy
RNA, Messenger
Rabbits
Receptors, Cytokine
Receptors, HIV
Recombinant Proteins
Retinal Neovascularization
Retinal Vessels
Reverse Transcriptase Polymerase Chain Reaction
Umbilical Veins
Vitreous Body

Chemicals

Angiogenesis Inducing Agents
CX3C Chemokine Receptor 1
Chemokine CX3CL1
RNA, Messenger
Receptors, Cytokine
Receptors, HIV
Recombinant Proteins
Oxygen

Word Cloud

Created with Highcharts 10.0.0FKNBRECsassayvitreousPDRangiogenicendothelialHUVECsangiogenesisvitrovivoocularcapillarysamplespatientsinducedFractalkineroledisorderscellsretinalchemotaxissyntheticmatrixformationRT-PCRWesternblotanalysisusedCX3CR1rabbitcornealneovascularizationretinopathyOIRmodelImmunodepletionantibodycellsignificantlymigrationcontrollevelelevatedmediatorPURPOSE:chemoattractantadhesionmoleculeleukocytesAngiogeniceffectalsoreportedstudyinvestigationFKN-mediateddetermineMETHODS:effectsculturedhumanumbilicalveinbovineevaluatedtubeReversetranscription-polymerasechainreactiondetectmRNAproteinexpressionreceptoroxygen-inducedmicetestpropertylevelsproliferativediabeticmeasuredenzyme-linkedimmunosorbentELISAanti-FKNpolyclonalobservedassaysRESULTS:tubesExpressiondetectedbloodvesselgrowthpocketIntravitrealinjectionanti-mousedecreasedcomparedsubjectssolublecaused366%lessCONCLUSIONS:mayplayimportantCX3Cchemokine

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