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Open Library of Bioscience

FAM3A As a Potential Molecular Target to Regulate Mitochondrial Respiration and ATP Production in Ischemic Heart.

Abstract

No abstract text available.

References

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3. Zhang X, et al. FAM3 gene family: a promising therapeutical target for NAFLD and type 2 diabetes. Metabolism. 2018;81:71–82. [DOI: 10.1016/j.metabol.2017.12.001]
4. Yan H, et al. FAM3A maintains metabolic homeostasis by interacting with F1-ATP synthase to regulate the activity and assembly of ATP synthase. Metabolism. 2023;139: 155372. [DOI: 10.1016/j.metabol.2022.155372]
5. Xu T, et al. FAM3A deficiency - induced mitochondrial dysfunction underlies post-infarct mortality and heart failure. J Cardiovasc Transl Res. 2023. https://doi.org/10.1007/s12265-023-10382-w . [DOI: 10.1007/s12265-023-10382-w]

MeSH Term

Chemicals