Difference between revisions of "LINC00319"
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LncBook ID: [https://bigd.big.ac.cn/lncbook/transcript?transid=HSALNT0289323 HSALNT0289323] | LncBook ID: [https://bigd.big.ac.cn/lncbook/transcript?transid=HSALNT0289323 HSALNT0289323] | ||
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===Function=== | ===Function=== | ||
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Biological Process: pathogenic process | Biological Process: pathogenic process | ||
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===Characteristics === | ===Characteristics === | ||
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[[File:Model of hLarp7 recognition of the 7SK.png|right|thumb|Model of hLarp7 recognition of the 7SK 3′end and mechanism of assembly of core 7SK RNP([https://www.ncbi.nlm.nih.gov/pubmed/29946027 (Eichhorn 2018)])]] | [[File:Model of hLarp7 recognition of the 7SK.png|right|thumb|Model of hLarp7 recognition of the 7SK 3′end and mechanism of assembly of core 7SK RNP([https://www.ncbi.nlm.nih.gov/pubmed/29946027 (Eichhorn 2018)])]] | ||
+ | Linc00319 promotes cell proliferation and invasion in lung cancer cells by directly binding with and downregulating the tumor suppressor miR-32.<ref name="ref1" /> | ||
===Disease=== | ===Disease=== | ||
− | + | lung cancer <ref name="ref1" /> | |
===Evolution=== | ===Evolution=== |
Revision as of 05:19, 12 August 2019
Contents
Annotation
Name
Approved symbol: LINC00319
Approved name: long intergenic non-protein coding RNA 319
HGNC ID: HGNC:19730
Previous symbols: C21orf125; NCRNA00319
Previous names: chromosome 21 open reading frame 125; non-protein coding RNA 319
Alias symbols: PRED49; FLJ38036
RefSeq ID: NR_026960
LncBook ID: HSALNT0289323
Function
Function Mechanism: ceRNA (PMID:28800794)
Biological Process: pathogenic process
Characteristics
Expression
Regulation
Function
Linc00319 promotes cell proliferation and invasion in lung cancer cells by directly binding with and downregulating the tumor suppressor miR-32.[1]
Disease
lung cancer [1]
Evolution
Please input evolution information here.
Labs working on this lncRNA
- College of Veterinary Medicine, Northwest A&F University, Yangling, Shaanxi 712100, China.[1]