OMIX008264

1Summary
Title Identification of glutamine as a potential therapeutic target in dry eye disease
Description Herein, we proposed a novel approach to modulate the inflammation through metabolic remodeling, thus promoting dry eye recovery. We demonstrated that co-treatment with MSCs and thymosin beta-4 (Tβ4) yielded the best therapeutic outcome against dry eye. Inhibition of glutamine reversed the anti-inflammatory, anti-apoptotic, and homeostasis-preserving effects observed with combined therapy, highlighting the critical role of glutamine in dry eye therapy. Clinical cases and rodent model showed elevated expression of glutaminase (GLS1), an upstream enzyme in glutamine metabolism, following dry eye injury. Mechanistic studies indicated that overexpression and inhibition of GLS1 counteracted and enhanced, respectively, the anti-inflammatory effects of combined therapy, underscoring GLS1's pivotal role in regulating glutamine metabolism.
Organism Rattus norvegicus
Data Type Expression Profiling
Data Accessibility Open-access
BioProject PRJCA033622
Release Date 2025-02-01
Submitter Min Zhang (mzhangcool@yeah.net)
Organization the First Medical Center of PLA General Hospital
Submission Date 2024-12-13
2Files & Download

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File ID File Title Number/Samples File Type File Size File Suffix Download
OMIX008264-01 DED 2 1 Expression Profiling 14.45 MB tar.gz
OMIX008264-02 Control 1 1 Expression Profiling 15.12 MB tar.gz
OMIX008264-03 GLS treatment 1 1 Expression Profiling 18.53 MB tar.gz
OMIX008264-04 GLS treatment 2 1 Expression Profiling 10.52 MB tar.gz
OMIX008264-05 GLS treatment 3 1 Expression Profiling 15.29 MB tar.gz
OMIX008264-06 Control 3 1 Expression Profiling 21.62 MB tar.gz
OMIX008264-07 Control 2 1 Expression Profiling 22.37 MB tar.gz
OMIX008264-08 DED 1 1 Expression Profiling 18.75 MB tar.gz
OMIX008264-09 DED 3 1 Expression Profiling 19.8 MB tar.gz
3Relevant Publications
Paper Title Journal Name Publish Time Accession Citing Type
Identification of glutamine as a potential therapeutic target in dry eye disease Signal Transduction and Targeted Therapy 2025-01 OMIX008264 Deposit

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