OMIX016920

1Summary
Title Mechanism of FLI1 in promoting glioblastoma development
Description Glioma is a common primary malignant brain tumor characterized by high malignancy and poor prognosis. Currently, there are no effective targeted therapies, making the identification of novel potential therapeutic targets an urgent priority. As a member of the ETS transcription factor family, FLI1 (Friend Leukemia Virus Integration 1) plays a pivotal role in the progression of various solid tumors. We observed that aberrant overexpression of FLI1 is closely associated with malignant phenotypes and poor prognosis in glioma. However, the specific role and underlying mechanisms of FLI1 in glioma progression remain unclear. This study investigates and validates the oncogenic role and mechanisms of FLI1 in glioma development. Using FLI1 Co-IP, we screened for and identified novel target molecules. Our experimental data demonstrate that FLI1 not only drives tumorigenesis through canonical oncogenic pathways but also regulates glioma progression across multiple levels.
Organism Homo sapiens
Data Type Proteomic Data by Mass Spectrometry (MS)
Data Accessibility Open-access
BioProject PRJCA063963
Release Date 2026-05-15
Submitter Jifan Hu (wenxuejdyy@jlu.edu.cn)
Organization First Hospital of Jilin University
Submission Date 2026-05-14
2Files & Download

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File ID File Title Number/Samples File Type File Size File Suffix Download
OMIX016920-01 U87MG-IgG-CoIP 1 Proteomic Data by Mass Spectrometry (MS) 531.22 MB raw
OMIX016920-02 U251MG-FLI1-CoIP 1 Proteomic Data by Mass Spectrometry (MS) 534.77 MB raw
OMIX016920-03 U87MG-FLI1-CoIP 1 Proteomic Data by Mass Spectrometry (MS) 515.2 MB raw
OMIX016920-04 U251MG-IgG-CoIP 1 Proteomic Data by Mass Spectrometry (MS) 490.27 MB raw
3Relevant Publications
Paper Title Journal Name Publish Time Accession Citing Type
FLI1 enhances the malignant phenotype of glioma cells and exerts immunomodulatory effects through feedback crosstalk with exonic circRNA FECR1 and interferon‑induced ISG15 International Journal of Molecular Medicine 2026-07 OMIX016920 Reference

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