Difference between revisions of "Os01g0689451"
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* The expression of iron deficiency-inducible genes involved in iron utilization is enhanced in OsHRZ-knockdown plants, mostly under iron-sufficient conditions. | * The expression of iron deficiency-inducible genes involved in iron utilization is enhanced in OsHRZ-knockdown plants, mostly under iron-sufficient conditions. | ||
* '''''OsHRZ1''''' is iron-binding sensors that may negatively regulate iron acquisition under conditions of iron sufficiency. | * '''''OsHRZ1''''' is iron-binding sensors that may negatively regulate iron acquisition under conditions of iron sufficiency. | ||
| + | * '''''OsHRZ1''''' is Fe- and Zn-binding ubiquitin ligases. | ||
===Expression=== | ===Expression=== | ||
Revision as of 02:24, 10 October 2016
The rice Os01g0689451 was reported as OsHRZ1 in 2013 [1] by researchers from Japan.
Contents
Annotated Information
Gene Symbol
- Os01g0689451 <=> OsHRZ1,HRZ1
Function
- OsHRZ1 is susceptible to degradation in roots irrespective of iron conditions.
- The expression of iron deficiency-inducible genes involved in iron utilization is enhanced in OsHRZ-knockdown plants, mostly under iron-sufficient conditions.
- OsHRZ1 is iron-binding sensors that may negatively regulate iron acquisition under conditions of iron sufficiency.
- OsHRZ1 is Fe- and Zn-binding ubiquitin ligases.
Expression
Please input expression information here.
Evolution
Please input evolution information here.
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Labs working on this gene
- Japan Science and Technology Agency, PRESTO, 4-1-8 Honcho, Kawaguchi, Saitama 332-0012, Japan.
- Research Institute for Bioresources and Biotechnology, Ishikawa Prefectural University, 1-308 Suematsu, Nonoichi, Ishikawa 921-8836, Japan.
- Graduate School of Agricultural and Life Sciences, The University of Tokyo, 1-1-1 Yayoi, Bunkyo-ku, Tokyo 113-8657, Japan.
References
- ↑ Kobayashi T, Nagasaka S, Senoura T, Itai RN, Nakanishi H, Nishizawa NK. Iron-binding haemerythrin RING ubiquitin ligases regulate plant iron responses and accumulation. Nat Commun. 2013;4:2792. doi: 10.1038/ncomms3792. PubMed PMID: 24253678;