OMIX012702

1Summary
Title CM-TAMs regulates TBLCs via BACH1/ANGPTL4/SDC4
Description This study reveals that tumor budding-like structures are common in chordomas and are closely associated with poor prognosis. Through multi-omics analysis and large-scale cohort validation, researchers identified a tumor budding-like cell subpopulation and uncovered its interaction with cholesterol metabolism tumor-associated macrophages. Mechanistically, BACH1-driven cholesterol metabolism tumor-associated macrophages secrete ANGPTL4, which activates tumor budding-like cells through SDC4, promoting their stemness, cholesterol accumulation, and malignant progression. In terms of treatment, inhibiting cholesterol metabolism or blocking the BACH1\ANGPTL4\SDC4 axis can suppress tumor invasiveness in preclinical models. Overall, this work redefines tumor budding-like structures as a key prognostic and mechanistic feature of chordomas and identifies a new immunometabolic signaling axis, providing potential therapeutic targets.
Organism Homo sapiens
Data Type Expression Profiling
Data Accessibility Controlled-access
BioProject PRJCA049985
Release Date 2026-01-02
Submitter Mingxiang Zou (zoumx_spine@usc.edu.cn)
Organization First affiliated hospital of university of South China
Submission Date 2025-11-01
2Files & Download

The data cannot be downloaded as it has not yet been registered in the Human Genetic Resource Management Platform of MOST.

File ID File Title Number/Samples File Type File Size File Suffix Download
OMIX012702-01 scATAC 3 Expression Profiling 224.4 MB zip Unavailable
OMIX012702-02 bulk-RNA seq 126 Expression Profiling 19.2 MB zip Unavailable
OMIX012702-03 scRNA-seq of CH 9 Expression Profiling 662.8 MB zip Unavailable
OMIX012702-04 scRNA-seq of IVD 5 Expression Profiling 52.8 MB zip Unavailable
3Relevant Publications
Paper Title Journal Name Publish Time Accession Citing Type
Cholesterol-metabolic TAMs regulates tumor budding like cell subpopulation to promote chordoma stemness via BACH1/ANGPTL4/SDC4 axis Neuro-oncology 2025-12 OMIX012702 Secondary use

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