OMIX008219

1Summary
Title Metabolic Recoding of 5'-Methylthioadenosine- Mediated H3K79me1 Demethylation Antagonizes Cadmium-Induced Breast Cancer Progression by Maintaining Autophagic Flux
Description For LC-MS analysis, a precooled methanol-water solution was utilized for the extraction of cellular metabolites, and the subsequent mass spectrometric information was acquired using a Nexera UPLC coupled to a Q Exactive high-resolution mass spectrometer. GC-MS analysis was conducted on a 7890B-5977A GC-MS system (Agilent Technologies, Santa Clara, CA, USA) equipped with a DB-5MS column, employing a temperature gradient and electron ionization (EI) for metabolite separation and detection.
Organism Homo sapiens
Data Type Metabolome Data by Mass Spectrometry (MS)
Data Accessibility Open-access
BioProject PRJCA033456
Release Date 2026-01-03
Submitter li li (laoweishuju@163.com)
Organization Third Military Medical University(Army Medical University)
Submission Date 2024-12-10
2Files & Download

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File ID File Title Number/Samples File Type File Size File Suffix Download
OMIX008219-01 raw data 15 Metabolome Data by Mass Spectrometry (MS) 1.94 GB zip
3Relevant Publications
Paper Title Journal Name Publish Time Accession Citing Type
5’‐Methylthioadenosine Metabolic Reprogramming Drives H3K79 Monomethylation‐Mediated PAK2 Upregulation to Promote Cadmium‐Induced Breast Cancer Progression by Impairing Autophagic Flux Advanced Science 2025-09 OMIX008223 OMIX008219 Deposit

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