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| Mutant name | PSR26r |
| Mutant/Transgenic |
transgenic |
| Ecotype |
Japanese morning glory (Ipomoea nil) |
| Mutagenesis type |
RNAi |
| (Semi-)Dominant/Recessive |
recessive |
| Description |
The deduced InPSR26 peptide consists of 152 amino acids and shows 50% amino acid sequence identity over the length of the entire peptide with putative membrane proteins of Arabidopsis (At4g15610 and At3g06390), which have unknown function. |
| References |
1: Shibuya K, Yamada T, Suzuki T, Shimizu K, Ichimura KInPSR26, a putative membrane protein, regulates programmed cell death during petal senescence in Japanese morning glory.Plant Physiol. 2009 Feb;149(2):816-24
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| | GenBank ID |
AB267825 | | Alias |
InPSR26 | | Organism |
Japanese morning glory (Ipomoea nil) | | Description |
InPSR26 is dominantly expressed in petal limbs and its transcript level increases prior to visible senescence symptoms. Transgenic plants with reduced InPSR26 expression (PSR26r lines) showed accelerated petal wilting, with PCD symptoms including cell collapse, ion and anthocyanin leakage, and DNA degradation accelerated in petals compared to wild-type plants. Transcript levels of autophagy- and PCD-related genes (InATG4, InATG8, InVPE, and InBI-1) were reduced in the petals of PSR26r plants. InPSR26 acts to delay the progression of PCD during petal senescence, possibly through regulation of the autophagic process. |
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