Basic information   
GenBank ID AB267825
AliasInPSR26
OrganismJapanese morning glory (Ipomoea nil)
Taxonomic identifier[NCBI]
Function categoryLipid/Carbohydrate metabolism:Macromolecule degradation
Effect for Senescencedelay
Gene DescriptionInPSR26 is dominantly expressed in petal limbs and its transcript level increases prior to visible senescence symptoms. Transgenic plants with reduced InPSR26 expression (PSR26r lines) showed accelerated petal wilting, with PCD symptoms including cell collapse, ion and anthocyanin leakage, and DNA degradation accelerated in petals compared to wild-type plants. Transcript levels of autophagy- and PCD-related genes (InATG4, InATG8, InVPE, and InBI-1) were reduced in the petals of PSR26r plants. InPSR26 acts to delay the progression of PCD during petal senescence, possibly through regulation of the autophagic process.
EvidenceGenetic evidence:Transgene as well as Molecular evidence: Northern [Ref 1]
References
1: Shibuya K, Yamada T, Suzuki T, Shimizu K, Ichimura K
InPSR26, a putative membrane protein, regulates programmed cell death during petal senescence in Japanese morning glory.
Plant Physiol. 2009 Feb;149(2):816-24

SequenceAB267825.2 | mRNA | CDS | Protein
Mutant information   
Mutant name PSR26r
Mutant/Transgenic transgenic
Ecotype Japanese morning glory (Ipomoea nil)
Mutagenesis type RNAi
Cross Link      
DatabaseEntry IDE-valueStartEndInterPro IDDescription
PANTHERPTHR116152.6E-341148No hitNA
PfamPF045354.0E-321134IPR006702Uncharacterised protein family UPF0497, trans-membrane plant
TIGRFAMTIGR015692.1E-371145IPR006459Uncharacterised protein family UPF0497, trans-membrane plant subgroup