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| Mutant name | spl28 |
| Mutant/Transgenic |
mutant |
| Ecotype |
Hwacheongbyeo (an elite Korean japonica cultivar) |
| Mutagenesis type |
N-methyl-N-nitrosourea (MNU) treatment |
| (Semi-)Dominant/Recessive |
recessive |
| Description |
Early stage development of the spl28 mutant was normal. However, after flowering, spl28 mutants exhibited a significant decrease in chlorophyll content, soluble protein content, and photosystem II efficiency, and high concentrations of reactive oxygen species (ROS), phytoalexin, callose, and autofluorescent phenolic compounds that localized in or around the lesions. The spl28 mutant also exhibited significantly enhanced resistance to rice blast and bacterial blight. |
| References |
1: Qiao Y, Jiang W, Lee J, Park B, Choi MS, Piao R, Woo MO, Roh JH, Han L, Paek NC, Seo HS, Koh HJSPL28 encodes a clathrin-associated adaptor protein complex 1, medium subunit micro 1 (AP1M1) and is responsible for spotted leaf and early senescence in rice (Oryza sativa).New Phytol. 2010 Jan;185(1):258-74
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| | Locus name |
LOC_Os01g50770 | | Alias |
SPL28 | | Organism |
Rice (Oryza sativa) | | Description |
SPL28 encodes a clathrin-associated adaptor protein complex 1, medium subunit μ1 (AP1M1), which is involved in the post-Golgi trafficking pathway. A green fluorescent protein (GFP) fusion protein of SPL28 (SPL28::GFP) localized to the Golgi apparatus. |
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