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| GenBank ID | AF134835 |
| Alias | CCS52A | | Organism | Alfalfa (Medicago sativa) | | Taxonomic identifier | [NCBI] | | Function category | Signal transduction:Cell cycle regulation | | Effect for Senescence | delay | | Gene Description | The CCS52A protein was present in the nucleus of endoreduplication-competent cells, indicating that it may activate APC constitutively during the endoreduplication cycles. Downregulation of ccs52A in transgenic M. truncatula plants drastically affected nodule development, resulting in lower ploidy, reduced cell size, inefficient invasion, and the maturation of symbiotic cells, accompanied by early senescence and finally the death of both the bacterium and plant cells. | | Evidence | Genetic evidence:Mutant [Ref 1] | | References | 1: Vinardell JM, Fedorova E, Cebolla A, Kevei Z, Horvath G, Kelemen Z, Tarayre S, Roudier F, Mergaert P, Kondorosi A, Kondorosi EEndoreduplication mediated by the anaphase-promoting complex activator CCS52A is required for symbiotic cell differentiation in Medicago truncatula nodules.Plant Cell 2003 Sep;15(9):2093-105 | | Gene Ontology | | | Sequence | AF134835.1 | Genomic | mRNA | CDS | Protein
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| | Mutant name |
CCS52A_RNAi | | Mutant/Transgenic |
transgenic | | Ecotype |
Medicago sativa |
| Mutagenesis type |
RNAi |
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