Megakaryocyte centrosomal and Golgi structural perturbations in patients with primary myelofibrosis and with germline mutation. - National Genomics Data Center (CNCB-NGDC)

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Megakaryocyte centrosomal and Golgi structural perturbations in patients with primary myelofibrosis and with germline mutation.

Ifeyinwa E Obiorah, Debamita Kundu, Folashade Adekunle, Patcharin Pramoonjago, Katherine R Calvo, Paul Liu, Adam Goldfarb

Author Information

Ifeyinwa E Obiorah: Department of Pathology, University of Virginia Health, Charlottesville, VA, USA.
Debamita Kundu: Department of Biostatistics, University of Virginia, Charlottesville, VA, USA.
Folashade Adekunle: Department of Pathology, University of Virginia Health, Charlottesville, VA, USA.
Patcharin Pramoonjago: Department of Pathology, University of Virginia Health, Charlottesville, VA, USA.
Katherine R Calvo: Department of Laboratory Medicine, National Institutes of Health Clinical Center, NIH, Bethesda, MD, USA.
Paul Liu: Genetics and Molecular Biology Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, MD, USA.
Adam Goldfarb: Department of Pathology, University of Virginia Health, Charlottesville, VA, USA.

PMID: 39654392 DOI: 10.1080/10428194.2024.2438801

No abstract text available.

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R01 HL149667/NHLBI NIH HHS

Journal Article

A patient with a germline GATA2 mutation and primary myelofibrosis.Megakaryocyte morphology with GATA2 germline mutation.Megakaryocyte dysplasia in primary myelofibrosis.Calreticulin mutation profile in Indian patients with primary myelofibrosis.[Primary myelofibrosis with double mutation in U2AF1].Characteristics of circulating megakaryocyte progenitors (CFU-MK) in patients with primary myelofibrosis.Abnormal megakaryocyte release and myelofibrosis.[JAK2V617F mutation in patients with idiopathic myelofibrosis].Circulating megakaryocyte in primary myelofibrosis: an uncommon finding in a myelofibrosis blood smear.[Fibrosis-driving cells in patients with primary myelofibrosis and myelodysplastic syndromes with myelofibrosis].

No available data.