PPGR - Gene Detail

Gene ID	Pop_G19G034324 View in Genome Browser
Position	chrG19:15711919-15716534 (-) 4615bp
Gene Type	gene
Gene Description (Protein Product)	Belongs to the disease resistance NB-LRR family
Organism	Populus alba x Populus glandulosa
Also AS	Potri.T023966AT4G27220Potri.T023966.v4.1

upstream:

Database	EntryID	E-Value	Start	end	InterPro ID	Description

Protein-protein interaction (PPI)	Pop_G19G080795 Belongs to the disease resistance NB-LRR family Pop_UnG033594 Belongs to the disease resistance NB-LRR family Pop_G19G080785 Belongs to the disease resistance NB-LRR family
Regulatory gene	Pop_A01G003924 Zinc finger protein Pop_A01G004096 transcriptional regulator Pop_A01G004283 ZINC FINGER protein

Load All Networks

Orthologous ID	Species Number	All hits in PereRegDB	Hits of this species	Orthologous Detail

DataSet	Number of Samples expressed(TPM>1)	Mean	Min	Max	Standard deviation(SD)	Coeffcient variation(CV)

Select Expression

GO Term	Description	GO Category
GO:0005575	cellular_component	CC
GO:0005623	obsolete cell	CC
GO:0005886	plasma membrane	CC
GO:0006950	response to stress	BP
GO:0006952	defense response	BP
GO:0007154	cell communication	BP
GO:0007165	signal transduction	BP
GO:0008150	biological_process	BP
GO:0009605	response to external stimulus	BP
GO:0009607	response to biotic stimulus	BP
GO:0009617	response to bacterium	BP
GO:0009987	cellular process	BP
GO:0016020	membrane	CC
GO:0023052	signaling	BP
GO:0043207	response to external biotic stimulus	BP
GO:0044464	obsolete cell part	CC
GO:0050789	regulation of biological process	BP
GO:0050794	regulation of cellular process	BP
GO:0050896	response to stimulus	BP
GO:0051704	obsolete multi-organism process	BP
GO:0051707	response to other organism	BP
GO:0051716	cellular response to stimulus	BP
GO:0065007	biological regulation	BP
GO:0071944	cell periphery	CC

KEGG Term	Name	Description
map04626	Plant-pathogen interaction	Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.