Basic Information
Gene Structure
Domain
Regulation&
Interaction
Annotation
Orthologus Group
Pathway
Basic Information
Gene ID
EVM0027039
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Position
GWHASIS00000114:18692968-18696595 (-)
3627bp
Gene Type
gene
Gene Description (Protein Product)
RPM1-interacting protein
Organism
Acer catalpifolium
Also AS AT3G25070

Gene Structure

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Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
EVM0032593 Belongs to the disease resistance NB-LRR family
EVM0028913 Serine threonine-protein kinase
EVM0029008 Belongs to the disease resistance NB-LRR family
Regulatory gene
EVM0001315 transcription factor that promotes early floral meristem identity in synergy with APETALA1; FRUITFULL and LEAFY. Is required subsequently for the transition of an inflorescence meristem into a floral meristem. Seems to be partially redundant to the function of APETALA1
EVM0003629 MADS-box protein
EVM0005245 MADS-box protein

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail


Pathway
Go Pathway KEGG Pathway
GO Term Description GO Category
GO:0002218 activation of innate immune response BP
GO:0002237 response to molecule of bacterial origin BP
GO:0002253 activation of immune response BP
GO:0002376 immune system process BP
GO:0002682 regulation of immune system process BP
GO:0002683 negative regulation of immune system process BP
GO:0002684 positive regulation of immune system process BP
GO:0005575 cellular_component CC
GO:0005623 obsolete cell CC
GO:0005886 plasma membrane CC
GO:0006464 protein modification process BP
GO:0006468 protein phosphorylation BP
GO:0006793 phosphorus metabolic process BP
GO:0006796 phosphate-containing compound metabolic process BP
GO:0006807 nitrogen compound metabolic process BP
GO:0006950 response to stress BP
GO:0006952 defense response BP
GO:0006955 immune response BP
GO:0007154 cell communication BP
GO:0007165 signal transduction BP
GO:0008150 biological_process BP
GO:0008152 metabolic process BP
GO:0008219 cell death BP
GO:0009605 response to external stimulus BP
GO:0009607 response to biotic stimulus BP
GO:0009617 response to bacterium BP
GO:0009626 plant-type hypersensitive response BP
GO:0009814 defense response to other organism BP
GO:0009816 defense response to bacterium BP
GO:0009987 cellular process BP
GO:0010033 response to organic substance BP
GO:0010204 innate immune response-activating signaling pathway BP
GO:0010363 regulation of plant-type hypersensitive response BP
GO:0010941 regulation of cell death BP
GO:0012501 programmed cell death BP
GO:0016020 membrane CC
GO:0016310 phosphorylation BP
GO:0019538 protein metabolic process BP
GO:0019897 extrinsic component of plasma membrane CC
GO:0019898 extrinsic component of membrane CC
GO:0023052 signaling BP
GO:0031347 regulation of defense response BP
GO:0031348 negative regulation of defense response BP
GO:0031349 positive regulation of defense response BP
GO:0033554 cellular response to stress BP
GO:0034050 programmed cell death induced by symbiont BP
GO:0034051 negative regulation of plant-type hypersensitive response BP
GO:0036211 protein modification process BP
GO:0042221 response to chemical BP
GO:0042742 defense response to bacterium BP
GO:0043067 regulation of programmed cell death BP
GO:0043069 negative regulation of programmed cell death BP
GO:0043170 macromolecule metabolic process BP
GO:0043207 response to external biotic stimulus BP
GO:0043412 macromolecule modification BP
GO:0044237 cellular metabolic process BP
GO:0044238 primary metabolic process BP
GO:0044260 cellular macromolecule metabolic process BP
GO:0044267 protein metabolic process BP
GO:0044425 obsolete membrane part CC
GO:0044459 obsolete plasma membrane part CC
GO:0044464 obsolete cell part CC
GO:0045087 innate immune response BP
GO:0045088 regulation of innate immune response BP
GO:0045089 positive regulation of innate immune response BP
GO:0045824 negative regulation of innate immune response BP
GO:0048518 positive regulation of biological process BP
GO:0048519 negative regulation of biological process BP
GO:0048523 negative regulation of cellular process BP
GO:0048583 regulation of response to stimulus BP
GO:0048584 positive regulation of response to stimulus BP
GO:0048585 negative regulation of response to stimulus BP
GO:0050776 regulation of immune response BP
GO:0050777 negative regulation of immune response BP
GO:0050778 positive regulation of immune response BP
GO:0050789 regulation of biological process BP
GO:0050794 regulation of cellular process BP
GO:0050896 response to stimulus BP
GO:0051704 obsolete multi-organism process BP
GO:0051707 response to other organism BP
GO:0051716 cellular response to stimulus BP
GO:0060548 negative regulation of cell death BP
GO:0065007 biological regulation BP
GO:0071704 organic substance metabolic process BP
GO:0071944 cell periphery CC
GO:0080134 regulation of response to stress BP
GO:0080135 regulation of cellular response to stress BP
GO:0098542 defense response to other organism BP
GO:1901564 organonitrogen compound metabolic process BP
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.