Basic Information
Gene ID
Position
scaffold1571_size99543:34885-36541 (-)
1656bp
Gene Type
gene
Gene Description (Protein Product)
Calcium-binding protein
Organism
Also AS AT3G43810

Gene Structure

upstream:

Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
FSB0134905 Calcium-dependent protein kinase
FSB0128956 calcium-dependent protein kinase
FSB0122041 cyclic nucleotide-gated ion channel
Regulatory gene
FSB0100633 Protein BASIC PENTACYSTEINE6-like
FSB0104454 MADS-box protein
FSB0106101 Transcription factor

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail

Expression Profile
DataSet Number of Samples expressed(TPM>1) Mean Min Max Standard deviation(SD) Coeffcient variation(CV)


Pathway
GO Term Description GO Category
GO:0003674 molecular_function MF
GO:0005488 binding MF
GO:0005509 calcium ion binding MF
GO:0005515 protein binding MF
GO:0005575 cellular_component CC
GO:0005622 intracellular anatomical structure CC
GO:0005623 obsolete cell CC
GO:0005737 cytoplasm CC
GO:0019904 protein domain specific binding MF
GO:0043167 ion binding MF
GO:0043169 cation binding MF
GO:0044424 obsolete intracellular part CC
GO:0044464 obsolete cell part CC
GO:0046872 metal ion binding MF
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.
map04070 Phosphatidylinositol signaling system -