Basic Information
Gene Structure
Domain
Regulation&
Interaction
Annotation
Orthologus Group
Pathway
Basic Information
Gene ID
JhiChr11G10952.g
View in Genome Browser
Position
chr11:13188952-13195639 (-)
6687bp
Gene Type
gene
Gene Description (Protein Product)
respiratory burst oxidase
Organism
Juglans hindsii
Also AS AT5G51060

Gene Structure

upstream:
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Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
JhiChr15G12264.g Calcium-dependent protein kinase
JhiChr11G12041.g Calcium-dependent protein kinase
JhiChr12G11140.g Calcium-dependent protein kinase
Regulatory gene
JhiChr01G10098.g AP2-like ethylene-responsive transcription factor
JhiChr01G10351.g Tesmin/TSO1-like CXC domain
JhiChr01G10507.g AP2-like ethylene-responsive transcription factor

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail


Pathway
Go Pathway KEGG Pathway
GO Term Description GO Category
GO:0003674 molecular_function MF
GO:0003824 catalytic activity MF
GO:0005575 cellular_component CC
GO:0005623 obsolete cell CC
GO:0005886 plasma membrane CC
GO:0005887 plasma membrane CC
GO:0006091 generation of precursor metabolites and energy BP
GO:0007275 multicellular organism development BP
GO:0007584 response to nutrient BP
GO:0008150 biological_process BP
GO:0008152 metabolic process BP
GO:0009060 aerobic respiration BP
GO:0009605 response to external stimulus BP
GO:0009653 anatomical structure morphogenesis BP
GO:0009888 tissue development BP
GO:0009987 cellular process BP
GO:0009991 response to extracellular stimulus BP
GO:0010015 root morphogenesis BP
GO:0010033 response to organic substance BP
GO:0010053 root epidermal cell differentiation BP
GO:0010243 response to organonitrogen compound BP
GO:0014070 response to organic cyclic compound BP
GO:0014074 response to purine-containing compound BP
GO:0015980 energy derivation by oxidation of organic compounds BP
GO:0016020 membrane CC
GO:0016021 membrane CC
GO:0016174 NAD(P)H oxidase H2O2-forming activity MF
GO:0016491 oxidoreductase activity MF
GO:0016651 oxidoreductase activity, acting on NAD(P)H MF
GO:0022622 root system development BP
GO:0030154 cell differentiation BP
GO:0031224 obsolete intrinsic component of membrane CC
GO:0031226 obsolete intrinsic component of plasma membrane CC
GO:0031667 response to nutrient levels BP
GO:0032501 multicellular organismal process BP
GO:0032502 developmental process BP
GO:0033198 response to ATP BP
GO:0042221 response to chemical BP
GO:0042493 response to xenobiotic stimulus BP
GO:0044237 cellular metabolic process BP
GO:0044425 obsolete membrane part CC
GO:0044459 obsolete plasma membrane part CC
GO:0044464 obsolete cell part CC
GO:0045333 cellular respiration BP
GO:0046683 response to organophosphorus BP
GO:0048364 root development BP
GO:0048731 system development BP
GO:0048856 anatomical structure development BP
GO:0048869 cellular developmental process BP
GO:0050664 oxidoreductase activity, acting on NAD(P)H, oxygen as acceptor MF
GO:0050896 response to stimulus BP
GO:0055114 obsolete oxidation-reduction process BP
GO:0071944 cell periphery CC
GO:0090558 plant epidermis development BP
GO:0090627 plant epidermal cell differentiation BP
GO:0099402 plant organ development BP
GO:1901698 response to nitrogen compound BP
GO:1901700 response to oxygen-containing compound BP
GO:1905392 plant organ morphogenesis BP
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.