Basic Information
Gene ID
JmiChr005G13044.g
Position
chr05:38800405-38801498 (-)
1093bp
Gene Type
gene
Gene Description (Protein Product)
Cyclic nucleotide-gated ion channel 1-like
Organism
Also AS AT5G53130

Gene Structure

upstream:

Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
JmiChr008G10222.g Plasma membrane ATPase
JmiChr007G12484.g plasma membrane ATPase
JmiChr15G12301.g ATPase 8, plasma
Regulatory gene
JmiChr001G10086.g Zinc-finger homeodomain protein
JmiChr001G10108.g atrl6,rl6,rsm3
JmiChr001G10125.g transcription factor

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail


Pathway
GO Term Description GO Category
GO:0005575 cellular_component CC
GO:0005623 obsolete cell CC
GO:0005886 plasma membrane CC
GO:0005887 plasma membrane CC
GO:0008150 biological_process BP
GO:0016020 membrane CC
GO:0016021 membrane CC
GO:0031224 obsolete intrinsic component of membrane CC
GO:0031226 obsolete intrinsic component of plasma membrane CC
GO:0042391 regulation of membrane potential BP
GO:0044425 obsolete membrane part CC
GO:0044459 obsolete plasma membrane part CC
GO:0044464 obsolete cell part CC
GO:0065007 biological regulation BP
GO:0065008 regulation of biological quality BP
GO:0071944 cell periphery CC
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.