PPGR - Gene Detail

Basic Information

Gene Structure

Domain

Regulation&
Interaction

Annotation

Orthologus Group

Expresssion

Pathway

Basic Information

Gene ID	Juni_Chr2.1813.g View in Genome Browser
Position	Chr2:29939238-29949166 (-) 9928bp
Gene Type	gene
Gene Description (Protein Product)	Respiratory burst oxidase homolog protein
Organism	Juglans nigra
Also AS	AT1G64060

upstream:

Domain

Database	EntryID	E-Value	Start	end	InterPro ID	Description

Regulation&Interaction

Protein-protein interaction (PPI)	Juni_Chr6.2260.g Calcium-dependent protein kinase Juni_Chr8.1656.g Calcium-dependent protein kinase Juni_Chr5.1422.g Calcium-dependent protein kinase
Regulatory gene	Juni_Chr11.1944.g Protein BASIC PENTACYSTEINE6-like Juni_Chr13.1575.g SNW SKI-interacting Juni_Chr13.1579.g Protein BASIC PENTACYSTEINE2-like

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Annotation

Orthologous Group

Orthologous ID	Species Number	All hits in PereRegDB	Hits of this species	Orthologous Detail

Expression Profile

DataSet	Number of Samples expressed(TPM>1)	Mean	Min	Max	Standard deviation(SD)	Coeffcient variation(CV)

Select Expression

Pathway

GO Term	Description	GO Category
GO:0000160	phosphorelay signal transduction system	BP
GO:0001101	response to acid chemical	BP
GO:0002252	immune effector process	BP
GO:0002376	immune system process	BP
GO:0002679	respiratory burst involved in defense response	BP
GO:0003674	molecular_function	MF
GO:0003824	catalytic activity	MF
GO:0005575	cellular_component	CC
GO:0005623	obsolete cell	CC
GO:0005886	plasma membrane	CC
GO:0006950	response to stress	BP
GO:0006952	defense response	BP
GO:0006970	response to osmotic stress	BP
GO:0007154	cell communication	BP
GO:0007165	signal transduction	BP
GO:0007231	osmosensory signaling pathway	BP
GO:0008150	biological_process	BP
GO:0008152	metabolic process	BP
GO:0009058	biosynthetic process	BP
GO:0009628	response to abiotic stimulus	BP
GO:0009719	response to endogenous stimulus	BP
GO:0009723	response to ethylene	BP
GO:0009725	response to hormone	BP
GO:0009737	response to abscisic acid	BP
GO:0009738	abscisic acid-activated signaling pathway	BP
GO:0009755	hormone-mediated signaling pathway	BP
GO:0009873	ethylene-activated signaling pathway	BP
GO:0009987	cellular process	BP
GO:0010033	response to organic substance	BP
GO:0010119	regulation of stomatal movement	BP
GO:0010941	regulation of cell death	BP
GO:0016020	membrane	CC
GO:0016174	NAD(P)H oxidase H2O2-forming activity	MF
GO:0016491	oxidoreductase activity	MF
GO:0016651	oxidoreductase activity, acting on NAD(P)H	MF
GO:0016999	antibiotic metabolic process	BP
GO:0017000	antibiotic biosynthetic process	BP
GO:0017144	xenobiotic metabolic process	BP
GO:0023052	signaling	BP
GO:0032870	cellular response to hormone stimulus	BP
GO:0033036	macromolecule localization	BP
GO:0033037	polysaccharide localization	BP
GO:0033500	carbohydrate homeostasis	BP
GO:0033554	cellular response to stress	BP
GO:0033993	response to lipid	BP
GO:0035556	intracellular signal transduction	BP
GO:0042221	response to chemical	BP
GO:0042592	homeostatic process	BP
GO:0042743	hydrogen peroxide metabolic process	BP
GO:0043067	regulation of programmed cell death	BP
GO:0043069	negative regulation of programmed cell death	BP
GO:0044237	cellular metabolic process	BP
GO:0044249	cellular biosynthetic process	BP
GO:0044464	obsolete cell part	CC
GO:0045730	respiratory burst	BP
GO:0048519	negative regulation of biological process	BP
GO:0048523	negative regulation of cellular process	BP
GO:0048878	chemical homeostasis	BP
GO:0050664	oxidoreductase activity, acting on NAD(P)H, oxygen as acceptor	MF
GO:0050665	hydrogen peroxide biosynthetic process	BP
GO:0050789	regulation of biological process	BP
GO:0050794	regulation of cellular process	BP
GO:0050896	response to stimulus	BP
GO:0051179	localization	BP
GO:0051186	obsolete cofactor metabolic process	BP
GO:0051188	obsolete cofactor biosynthetic process	BP
GO:0051716	cellular response to stimulus	BP
GO:0052542	defense response by callose deposition	BP
GO:0052545	callose localization	BP
GO:0055114	obsolete oxidation-reduction process	BP
GO:0060548	negative regulation of cell death	BP
GO:0065007	biological regulation	BP
GO:0065008	regulation of biological quality	BP
GO:0070887	cellular response to chemical stimulus	BP
GO:0071214	cellular response to abiotic stimulus	BP
GO:0071215	cellular response to abscisic acid stimulus	BP
GO:0071229	cellular response to acid chemical	BP
GO:0071310	cellular response to organic substance	BP
GO:0071369	cellular response to ethylene stimulus	BP
GO:0071396	cellular response to lipid	BP
GO:0071470	cellular response to osmotic stress	BP
GO:0071495	cellular response to endogenous stimulus	BP
GO:0071944	cell periphery	CC
GO:0072593	reactive oxygen species metabolic process	BP
GO:0097305	response to alcohol	BP
GO:0097306	cellular response to alcohol	BP
GO:0104004	cellular response to environmental stimulus	BP
GO:1901700	response to oxygen-containing compound	BP
GO:1901701	cellular response to oxygen-containing compound	BP
GO:1903409	reactive oxygen species biosynthetic process	BP

KEGG Term	Name	Description
map04626	Plant-pathogen interaction	Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.