Basic Information
Gene ID
MD16G1166800.v1.1
Position
Chr16:13890600-13892014 (+)
1414bp
Gene Type
gene
Gene Description (Protein Product)
3-ketoacyl-CoA synthase
Organism
Also AS MD16G1166800AT1G71160

Gene Structure

upstream:

Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
MD17G1034800.v1.1 Catalyzes the NAD( )-dependent oxidation of formate to carbon dioxide. Involved in the cell stress response
MD17G1011500.v1.1 Omega-hydroxypalmitate O-feruloyl

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail

Expression Profile
DataSet Number of Samples expressed(TPM>1) Mean Min Max Standard deviation(SD) Coeffcient variation(CV)


Pathway
GO Term Description GO Category
GO:0003674 molecular_function MF
GO:0003824 catalytic activity MF
GO:0004312 fatty acid synthase activity MF
GO:0006950 response to stress BP
GO:0008150 biological_process BP
GO:0009266 response to temperature stimulus BP
GO:0009314 response to radiation BP
GO:0009409 response to cold BP
GO:0009416 response to light stimulus BP
GO:0009628 response to abiotic stimulus BP
GO:0009922 fatty acid elongase activity MF
GO:0016740 transferase activity MF
GO:0016746 acyltransferase activity MF
GO:0016747 acyltransferase activity, transferring groups other than amino-acyl groups MF
GO:0050896 response to stimulus BP
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.
map01110 Biosynthesis of secondary metabolites -
map01100 Metabolic pathways -
map00062 Fatty acid elongation in mitochondria -