Basic Information
Gene ID
MD17G1257900.v1.1
Position
Chr17:31837313-31837777 (+)
464bp
Gene Type
gene
Gene Description (Protein Product)
calcium-binding protein
Organism
Also AS MD17G1257900AT1G18210

Gene Structure

upstream:

Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
MD17G1258300.v1.1 DNA repair protein
MD17G1267100.v1.1 pfkB family carbohydrate kinase
MD17G1259800.v1.1 valine--tRNA ligase

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail

Expression Profile
DataSet Number of Samples expressed(TPM>1) Mean Min Max Standard deviation(SD) Coeffcient variation(CV)


Pathway
GO Term Description GO Category
GO:0001505 regulation of neurotransmitter levels BP
GO:0003674 molecular_function MF
GO:0005488 binding MF
GO:0005509 calcium ion binding MF
GO:0008150 biological_process BP
GO:0009909 regulation of flower development BP
GO:0019222 regulation of metabolic process BP
GO:0031323 regulation of cellular metabolic process BP
GO:0043167 ion binding MF
GO:0043169 cation binding MF
GO:0046872 metal ion binding MF
GO:0048580 regulation of post-embryonic development BP
GO:0048831 regulation of shoot system development BP
GO:0050789 regulation of biological process BP
GO:0050793 regulation of developmental process BP
GO:0050794 regulation of cellular process BP
GO:0051171 regulation of nitrogen compound metabolic process BP
GO:0051239 regulation of multicellular organismal process BP
GO:0065007 biological regulation BP
GO:0065008 regulation of biological quality BP
GO:0080164 regulation of nitric oxide metabolic process BP
GO:2000026 regulation of multicellular organismal development BP
GO:2000241 regulation of reproductive process BP
GO:2000377 regulation of reactive oxygen species metabolic process BP
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.