Basic Information
Gene Structure
Domain
Regulation&
Interaction
Annotation
Orthologus Group
Pathway
Basic Information
Gene ID
Potrs000182g00239
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Position
Potrs000182:30466-32233 (+)
1767bp
Gene Type
gene
Gene Description (Protein Product)
WRKY transcription factor
Organism
Populus tremuloides
Also AS Potri.014G090300AT4G01250Potri.014G090300.v4.1

Gene Structure

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Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
Potrs008727g13000 WRKY transcription factor
Potrs007026g09663 WRKY transcription factor
Potrs013103g17484 WRKY transcription factor
Regulatory gene
Potrs000037g00005 methyltransferase At2g41040, chloroplastic-like
Potrs000108g00058 Protein TRANSPARENT TESTA
Potrs000176g35795 C2H2-type zinc finger
Target gene
Potrs000022g00016 Non-catalytic subunit of the queuine tRNA- ribosyltransferase (TGT) that catalyzes the base-exchange of a guanine (G) residue with queuine (Q) at position 34 (anticodon wobble position) in tRNAs with GU(N) anticodons (tRNA-Asp, -Asn, - His and -Tyr), resulting in the hypermodified nucleoside queuosine (7-(((4,5-cis-dihydroxy-2-cyclopenten-1-yl)amino)methyl)-7- deazaguanosine)
Potrs000025g33696
Potrs000039g35135

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail


Pathway
Go Pathway KEGG Pathway
GO Term Description GO Category
GO:0003674 molecular_function MF
GO:0003700 DNA-binding transcription factor activity MF
GO:0006355 regulation of DNA-templated transcription BP
GO:0007275 multicellular organism development BP
GO:0007568 aging BP
GO:0008150 biological_process BP
GO:0009889 regulation of biosynthetic process BP
GO:0010033 response to organic substance BP
GO:0010150 leaf senescence BP
GO:0010200 response to chitin BP
GO:0010243 response to organonitrogen compound BP
GO:0010468 regulation of gene expression BP
GO:0010556 regulation of macromolecule biosynthetic process BP
GO:0019219 regulation of nucleobase-containing compound metabolic process BP
GO:0019222 regulation of metabolic process BP
GO:0031323 regulation of cellular metabolic process BP
GO:0031326 regulation of cellular biosynthetic process BP
GO:0032501 multicellular organismal process BP
GO:0032502 developmental process BP
GO:0042221 response to chemical BP
GO:0042493 response to xenobiotic stimulus BP
GO:0048366 leaf development BP
GO:0048367 shoot system development BP
GO:0048731 system development BP
GO:0048827 phyllome development BP
GO:0048856 anatomical structure development BP
GO:0050789 regulation of biological process BP
GO:0050794 regulation of cellular process BP
GO:0050896 response to stimulus BP
GO:0051171 regulation of nitrogen compound metabolic process BP
GO:0051252 regulation of RNA metabolic process BP
GO:0060255 regulation of macromolecule metabolic process BP
GO:0065007 biological regulation BP
GO:0080090 regulation of primary metabolic process BP
GO:0090693 plant organ senescence BP
GO:0099402 plant organ development BP
GO:0140110 transcription regulator activity MF
GO:1901698 response to nitrogen compound BP
GO:1901700 response to oxygen-containing compound BP
GO:1903506 regulation of nucleic acid-templated transcription BP
GO:2000112 regulation of cellular macromolecule biosynthetic process BP
GO:2001141 regulation of RNA biosynthetic process BP
KEGG Term Name Description
map04626 Plant-pathogen interaction Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.