PPGR - Gene Detail

Gene ID	Potrs016627g19275 View in Genome Browser
Position	Potrs016627:965-1456 (-) 491bp
Gene Type	gene
Gene Description (Protein Product)	calcium-binding protein CML27
Organism	Populus tremuloides
Also AS	Potri.012G048200AT1G18210Potri.012G048200.v4.1

upstream:

Database	EntryID	E-Value	Start	end	InterPro ID	Description

Protein-protein interaction (PPI)	Potrs038202g34903 WRKY transcription factor Potrs041693g26204 calcium-binding protein Potrs017285g19625 heat shock
Regulatory gene	Potrs000984g01639 GAGA binding protein-like family Potrs000984g01640 GAGA binding protein-like family Potrs002956g04589 Protein BASIC PENTACYSTEINE4-like

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Orthologous ID	Species Number	All hits in PereRegDB	Hits of this species	Orthologous Detail

GO Term	Description	GO Category
GO:0003674	molecular_function	MF
GO:0005488	binding	MF
GO:0005509	calcium ion binding	MF
GO:0005575	cellular_component	CC
GO:0005622	intracellular anatomical structure	CC
GO:0005623	obsolete cell	CC
GO:0005737	cytoplasm	CC
GO:0005773	vacuole	CC
GO:0005886	plasma membrane	CC
GO:0016020	membrane	CC
GO:0043167	ion binding	MF
GO:0043169	cation binding	MF
GO:0043226	organelle	CC
GO:0043227	membrane-bounded organelle	CC
GO:0043229	intracellular organelle	CC
GO:0043231	intracellular membrane-bounded organelle	CC
GO:0044424	obsolete intracellular part	CC
GO:0044444	obsolete cytoplasmic part	CC
GO:0044464	obsolete cell part	CC
GO:0046872	metal ion binding	MF
GO:0071944	cell periphery	CC

KEGG Term	Name	Description
map04626	Plant-pathogen interaction	Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.