Basic Information
Gene Structure
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Domain
| Database | EntryID | E-Value | Start | end | InterPro ID | Description |
|---|
Regulation&Interaction
Annotation
Orthologous Group
| Orthologous ID | Species Number | All hits in PereRegDB | Hits of this species | Orthologous Detail |
|---|
Pathway
| GO Term | Description | GO Category |
|---|---|---|
| GO:0003008 | system process | BP |
| GO:0003674 | molecular_function | MF |
| GO:0005488 | binding | MF |
| GO:0005509 | calcium ion binding | MF |
| GO:0005515 | protein binding | MF |
| GO:0005575 | cellular_component | CC |
| GO:0005622 | intracellular anatomical structure | CC |
| GO:0005623 | obsolete cell | CC |
| GO:0005634 | nucleus | CC |
| GO:0005737 | cytoplasm | CC |
| GO:0005856 | cytoskeleton | CC |
| GO:0006928 | obsolete movement of cell or subcellular component | BP |
| GO:0007600 | sensory perception | BP |
| GO:0008092 | cytoskeletal protein binding | MF |
| GO:0008150 | biological_process | BP |
| GO:0009987 | cellular process | BP |
| GO:0010941 | regulation of cell death | BP |
| GO:0010942 | positive regulation of cell death | BP |
| GO:0015629 | actin cytoskeleton | CC |
| GO:0016459 | myosin complex | CC |
| GO:0016461 | unconventional myosin complex | CC |
| GO:0017022 | myosin binding | MF |
| GO:0019233 | sensory perception of pain | BP |
| GO:0030029 | actin filament-based process | BP |
| GO:0030048 | actin filament-based movement | BP |
| GO:0031476 | myosin VI complex | CC |
| GO:0032028 | myosin head/neck binding | MF |
| GO:0032036 | myosin heavy chain binding | MF |
| GO:0032501 | multicellular organismal process | BP |
| GO:0032991 | protein-containing complex | CC |
| GO:0042981 | regulation of apoptotic process | BP |
| GO:0043065 | positive regulation of apoptotic process | BP |
| GO:0043067 | regulation of programmed cell death | BP |
| GO:0043068 | positive regulation of programmed cell death | BP |
| GO:0043167 | ion binding | MF |
| GO:0043169 | cation binding | MF |
| GO:0043226 | organelle | CC |
| GO:0043227 | membrane-bounded organelle | CC |
| GO:0043228 | non-membrane-bounded organelle | CC |
| GO:0043229 | intracellular organelle | CC |
| GO:0043231 | intracellular membrane-bounded organelle | CC |
| GO:0043232 | intracellular non-membrane-bounded organelle | CC |
| GO:0044422 | obsolete organelle part | CC |
| GO:0044424 | obsolete intracellular part | CC |
| GO:0044430 | obsolete cytoskeletal part | CC |
| GO:0044446 | obsolete intracellular organelle part | CC |
| GO:0044464 | obsolete cell part | CC |
| GO:0046872 | metal ion binding | MF |
| GO:0048518 | positive regulation of biological process | BP |
| GO:0048522 | positive regulation of cellular process | BP |
| GO:0050789 | regulation of biological process | BP |
| GO:0050794 | regulation of cellular process | BP |
| GO:0050877 | nervous system process | BP |
| GO:0065007 | biological regulation | BP |
| GO:0070853 | myosin VI binding | MF |
| GO:0070854 | myosin VI heavy chain binding | MF |
| GO:0070855 | myosin VI head/neck binding | MF |
| GO:0070864 | sperm individualization complex | CC |
| GO:0070865 | investment cone | CC |
| KEGG Term | Name | Description |
|---|---|---|
| map04626 | Plant-pathogen interaction | Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance. |
| map04626 | Plant-pathogen interaction | Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance. |
| map04070 | Phosphatidylinositol signaling system | - |