Basic Information
Gene Structure
Domain
Regulation&
Interaction
Annotation
Orthologus Group
Expresssion
Pathway
Basic Information
Gene ID
PSME_50132.g
Position
jcf7190000009941:510108-648898 (-)
138790bp
Gene Type
gene
Gene Description (Protein Product)
DNA repair protein
Organism
Pseudotsuga menziesii
Also AS AT1G80420

Gene Structure

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Domain
Database EntryID E-Value Start end InterPro ID Description

Regulation&Interaction
Protein-protein interaction (PPI)
PSME_51375.g DNA mismatch repair protein
PSME_51318.g Component of the post-replicative DNA mismatch repair system (MMR)
PSME_51331.g Belongs to the cullin family
Regulatory gene
PSME_05476.g WUSCHEL-related homeobox
PSME_09831.g Heat stress transcription factor
PSME_09979.g Dof zinc finger protein

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Annotation

Orthologous Group
Orthologous ID Species Number All hits in PereRegDB Hits of this species Orthologous Detail

Expression Profile
DataSet Number of Samples expressed(TPM>1) Mean Min Max Standard deviation(SD) Coeffcient variation(CV)


Pathway
Go Pathway KEGG Pathway
GO Term Description GO Category
GO:0000726 obsolete non-recombinational repair BP
GO:0001666 response to hypoxia BP
GO:0003674 molecular_function MF
GO:0003676 nucleic acid binding MF
GO:0003677 DNA binding MF
GO:0003690 double-stranded DNA binding MF
GO:0005488 binding MF
GO:0005575 cellular_component CC
GO:0005622 intracellular anatomical structure CC
GO:0005623 obsolete cell CC
GO:0005634 nucleus CC
GO:0006139 nucleobase-containing compound metabolic process BP
GO:0006259 DNA metabolic process BP
GO:0006266 DNA ligation BP
GO:0006281 DNA repair BP
GO:0006284 base-excision repair BP
GO:0006302 double-strand break repair BP
GO:0006303 double-strand break repair via nonhomologous end joining BP
GO:0006304 DNA modification BP
GO:0006725 cellular aromatic compound metabolic process BP
GO:0006807 nitrogen compound metabolic process BP
GO:0006950 response to stress BP
GO:0006974 cellular response to DNA damage stimulus BP
GO:0008150 biological_process BP
GO:0008152 metabolic process BP
GO:0009628 response to abiotic stimulus BP
GO:0009893 positive regulation of metabolic process BP
GO:0009987 cellular process BP
GO:0010033 response to organic substance BP
GO:0010385 double-stranded methylated DNA binding MF
GO:0010562 positive regulation of phosphorus metabolic process BP
GO:0010604 positive regulation of macromolecule metabolic process BP
GO:0010921 regulation of phosphatase activity BP
GO:0010922 positive regulation of phosphatase activity BP
GO:0019219 regulation of nucleobase-containing compound metabolic process BP
GO:0019220 regulation of phosphate metabolic process BP
GO:0019222 regulation of metabolic process BP
GO:0031323 regulation of cellular metabolic process BP
GO:0031325 positive regulation of cellular metabolic process BP
GO:0033554 cellular response to stress BP
GO:0034641 cellular nitrogen compound metabolic process BP
GO:0035303 regulation of dephosphorylation BP
GO:0035306 positive regulation of dephosphorylation BP
GO:0035510 DNA dealkylation BP
GO:0036293 response to decreased oxygen levels BP
GO:0042221 response to chemical BP
GO:0042493 response to xenobiotic stimulus BP
GO:0043085 positive regulation of catalytic activity BP
GO:0043170 macromolecule metabolic process BP
GO:0043226 organelle CC
GO:0043227 membrane-bounded organelle CC
GO:0043229 intracellular organelle CC
GO:0043231 intracellular membrane-bounded organelle CC
GO:0043412 macromolecule modification BP
GO:0044093 positive regulation of molecular function BP
GO:0044237 cellular metabolic process BP
GO:0044238 primary metabolic process BP
GO:0044260 cellular macromolecule metabolic process BP
GO:0044424 obsolete intracellular part CC
GO:0044464 obsolete cell part CC
GO:0044728 obsolete DNA methylation or demethylation BP
GO:0045935 positive regulation of nucleobase-containing compound metabolic process BP
GO:0045937 positive regulation of phosphate metabolic process BP
GO:0046483 heterocycle metabolic process BP
GO:0048518 positive regulation of biological process BP
GO:0048522 positive regulation of cellular process BP
GO:0050789 regulation of biological process BP
GO:0050790 regulation of catalytic activity BP
GO:0050794 regulation of cellular process BP
GO:0050896 response to stimulus BP
GO:0051171 regulation of nitrogen compound metabolic process BP
GO:0051173 positive regulation of nitrogen compound metabolic process BP
GO:0051174 regulation of phosphorus metabolic process BP
GO:0051336 regulation of hydrolase activity BP
GO:0051345 positive regulation of hydrolase activity BP
GO:0051716 cellular response to stimulus BP
GO:0060255 regulation of macromolecule metabolic process BP
GO:0065007 biological regulation BP
GO:0065009 regulation of molecular function BP
GO:0070482 response to oxygen levels BP
GO:0070988 demethylation BP
GO:0071704 organic substance metabolic process BP
GO:0080090 regulation of primary metabolic process BP
GO:0080111 DNA demethylation BP
GO:0090304 nucleic acid metabolic process BP
GO:0097159 organic cyclic compound binding MF
GO:1901360 organic cyclic compound metabolic process BP
GO:1901363 heterocyclic compound binding MF
GO:1901968 regulation of polynucleotide 3'-phosphatase activity BP
GO:1901969 positive regulation of polynucleotide 3'-phosphatase activity BP
GO:1901971 regulation of DNA-5-methylcytosine glycosylase activity BP
GO:1901972 positive regulation of DNA-5-methylcytosine glycosylase activity BP
GO:1902544 regulation of DNA N-glycosylase activity BP
GO:1902546 positive regulation of DNA N-glycosylase activity BP
KEGG Term Name Description
map03410 Base excision repair Base excision repair (BER) is the predominant DNA damage repair pathway for the processing of small base lesions, derived from oxidation and alkylation damages. BER is normally defined as DNA repair initiated by lesion-specific DNA glycosylases and completed by either of the two sub-pathways: short-patch BER where only one nucleotide is replaced and long-patch BER where 2-13 nucleotides are replaced. Each sub-pathway of BER relies on the formation of protein complexes that assemble at the site of the DNA lesion and facilitate repair in a coordinated fashion. This process of complex formation appears to provide an increase in specificity and efficiency to the BER pathway, thereby facilitating the maintenance of genome integrity by preventing the accumulation of highly toxic repair intermediates.