PPGR - Gene Detail

Basic Information

Gene ID	gene-LOC112491970 View in Genome Browser
Position	NC_063292.1:15547374-15550055 (-) 2681bp
Gene Type	gene
Gene Description (Protein Product)	3-ketoacyl-CoA synthase
Organism	Ziziphus jujuba
Also AS	AT2G26640

upstream:

Domain

Database	EntryID	E-Value	Start	end	InterPro ID	Description

Regulation&Interaction

Protein-protein interaction (PPI)

gene-LOC112493419 Nucleoside diphosphate kinase

gene-LOC125418705 Adenylate isopentenyltransferase-like

gene-LOC125421195 Catalyzes the third of the four reactions of the long- chain fatty acids elongation cycle. This endoplasmic reticulum- bound enzymatic process; allows the addition of two carbons to the chain of long- and very long-chain fatty acids VLCFAs per cycle. This enzyme catalyzes the dehydration of the 3-hydroxyacyl-CoA intermediate into trans-2;3-enoyl-CoA; within each cycle of fatty acid elongation. Thereby; it participates to the production of VLCFAs of different chain lengths that are involved in multiple biological processes as precursors of membrane lipids and lipid mediators

Regulatory gene

gene-LOC107403899 dof zinc finger protein

gene-LOC107404240 Dof zinc finger protein

gene-LOC107406554 Dof domain; zinc finger

Load All Networks

Annotation

Orthologous Group

Orthologous ID	Species Number	All hits in PereRegDB	Hits of this species	Orthologous Detail

Expression Profile

DataSet	Number of Samples expressed(TPM>1)	Mean	Min	Max	Standard deviation(SD)	Coeffcient variation(CV)

Select Expression

Pathway

GO Term	Description	GO Category
GO:0006950	response to stress	BP
GO:0008150	biological_process	BP
GO:0009266	response to temperature stimulus	BP
GO:0009314	response to radiation	BP
GO:0009409	response to cold	BP
GO:0009416	response to light stimulus	BP
GO:0009628	response to abiotic stimulus	BP
GO:0050896	response to stimulus	BP

KEGG Term	Name	Description
map04626	Plant-pathogen interaction	Plants lack animal-like adaptive immunity mechanisms, and therefore have evolved a specific system with multiple layers against invading pathogens. The primary response includes the perception of pathogens by cell-surface pattern-recognition receptors (PRRs) and is referred to as PAMP-triggered immunity (PTI). Activation of FLS2 and EFR triggers MAPK signaling pathway that activates defense genes for antimictobial compounds. The increase in the cytosolic Ca2+ concentration is also a regulator for production of reactive oxygen species and localized programmed cell death/hypersensitive response. The secondary response is called effector-triggered immunity (ETI). Pathogens can acquire the ability to suppress PTI by directly injecting effector proteins into the plant cell through secretion systems. In addition, pathogens can manipulate plant hormone signaling pathways to evade host immune responses using coronatine toxin. Some plants possess specific intracellular surveillance proteins (R proteins) to monitor the presence of pathogen virulence proteins. This ETI occurs with localized programmed cell death to arrest pathogen growth, resulting in cultivar-specific disease resistance.
map01110	Biosynthesis of secondary metabolites	-
map01100	Metabolic pathways	-
map00062	Fatty acid elongation in mitochondria	-