bCCP1 - Plant Editosome Database

Editing Details

Species

Gene ID

Organelle

Edited Gene

Position

Region

Nuclear Genome

Organelle Genome

Other Position Region

Other Position

Editing Type

Codon

Amino Acid

Molecular Effect

Experiment Details

Zea mays

AC218148.2_FG008

Mitochondrion

atp4

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	94.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	76.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	46.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp4

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCA=>UUA

S=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	9.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	94.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	12.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp6

515

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCU=>UUU

S=>F

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	91.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp6

525

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	7.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	31.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp6

584

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	96.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	56.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	96.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	58.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp6

931

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	65.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp6

953

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCA=>UUA

S=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	63.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	81.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	7.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp8

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	4.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	24.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

atp9

228

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	65.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	17.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

172

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	86.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	82.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	67.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

181

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	86.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	58.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	78.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	62.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

193, 194

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCU=>UUU

P=>F

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	82%, 78%	High, High	None, None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	6%, 0%	Poor, Uned	Decreased,	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	73%, 70%	High, High	None, None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	35%, 0%	Medium, Un	Decreased,	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

392

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCG=>CUG

P=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	85.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	14.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	45.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

475, 476

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCA=>UUA

P=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80%, 76%	High, High	None, None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	47%, 3%	Medium, Po	Decreased,	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	76%, 73%	High, High	None, None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	54%, 6%	Medium, Po	Decreased,	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

485

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	38.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	76.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	50.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

503

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	46.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	76.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	48.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmB

566

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCC=>UUC

S=>F

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	5.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	9.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

-117

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	7.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

1144

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CUG=>UUG

L=>L

Synonymous

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	85.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	14.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

119

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	95.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

123

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	70.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	37.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

312

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	5.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

325

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	74.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	97.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

866

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	97.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	19.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFc

966

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCC=>CCU

P=>P

Synonymous

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	78.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1214

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	20.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	60.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	31.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1276

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	5.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	25.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1325

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCA=>CUA

P=>L

Synonymous

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1375

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	85.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	67.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	85.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1489

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	62.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	84.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

1553

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCU=>UUU

S=>F

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	81.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	19.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

181

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	62.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

287

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	62.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	72.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

514

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	35.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	59.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

595

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	86.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

752

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCA=>UUA

S=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	82.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	19.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	86.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	10.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	38.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

762

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	61.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

ccmFn

812

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	85.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	57.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	56.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cob

1098

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	54.04%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.85%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	40.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox1

1273

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

148

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	9.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	37.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

162

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

466

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	69.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	37.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

467

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	97.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	59.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	82.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	13.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

482

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	76.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	50.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	66.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

550

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	81.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

563

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	97.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	43.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	94.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	46.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox2

620

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCA=>UUA

S=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	84.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	17.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	20.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox3

566

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	50.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	70.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

cox3

657

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	9.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	29.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

1730

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	61.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	93.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

1751

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	60.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	63.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	24.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

1785

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	43.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

1885

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	30.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

1905

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	49.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	69.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

matR

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	50.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	73.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

mttB

127

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	17.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad1

215

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	98.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	55.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad1

537

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	50.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	93.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	43.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad1

571

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	91.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	51.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	98.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	66.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad1

835

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	16.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	40.00%	Medium	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	37.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

1057

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	22.00%	Low	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	14.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	43.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

1212

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

1247

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCA=>UUA

S=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	6.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

388

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	98.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	52.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	56.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

453

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	51.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad2

525

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	9.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad3

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad4

819

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	63.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	86.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad4

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	54.00%	Medium	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	21.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	44.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad4L

179

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	10.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	22.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	4.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad5

1490

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCC=>CUC

P=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	28.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	24.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad5

1494

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	54.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad5

1901

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	57.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	20.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad6

-3

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	24.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	37.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad6

191

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	96.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	46.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	96.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	79.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

1103

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

1124

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCA=>CUA

P=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	5.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

255

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	35.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	60.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

445

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	98.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	65.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	82.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

531

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	14.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

534

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	18.00%	Poor	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	45.00%	Medium	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

963

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	27.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	63.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	25.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	71.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad7

973

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCU=>UCU

P=>S

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	100.00%	Complete	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	99.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	4.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad9

111

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	26.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

nad9

298

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCG=>UCG

P=>S

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rpl16

184

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	87.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	62.00%	High	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	87.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	64.00%	High	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rpl16

228

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	42.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	74.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	59.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rpl16

287

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

ACA=>AUA

T=>I

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	23.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rpl16

444

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

UCC=>UCU

S=>S

Synonymous

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	57.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rpl16

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	71.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	13.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps1

377

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	68.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	25.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	73.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	41.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps12

289

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	35.00%	Low	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	61.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps13

256

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CGU=>UGU

R=>C

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	68.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	16.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps13

287

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	67.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	87.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps2A

449

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	87.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	39.00%	Low	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps2A

514

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	83.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	56.00%	Medium	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	80.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	53.00%	Medium	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps2B

455

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	77.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	2.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	89.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps3

1607

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	8.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	84.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	13.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps3

707

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	32.00%	Low	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	90.00%	High	Increased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	47.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	81.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps4

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CCA=>CUA

P=>L

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	67.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	79.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	0.00%	Unedited	Absent	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps4

482

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	55.00%	Medium	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	34.00%	Low	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	54.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	7.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps4

955

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

CAU=>UAU

H=>Y

Recoding

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
W22	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	69.00%	High	None	36200865
W22	bccp1-1	A Mu insertion	A Mu insertion 1,508-bp downstream from the translation start codon in AC218148.2_FG008 was identified to be linked with the bccp1-1 mutant in a six mutant and six wild-type family members.	Recessive; Homozygous	The mutant kernels at 14-day after pollination (DAP) were much smaller than the wild-type kernels. The mutant embryo was barely visible, and the endosperm was tiny. Paraffin sectioning showed that the wild-type kernels developed a complete embryonic structure at 14 DAP, whereas the bccp1-1 embryo only reached the transition stage, and the endosperm remained tiny.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	11.00%	Poor	Decreased	36200865
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	75.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	19.00%	Poor	Decreased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps7

-68

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	60.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	87.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps7

277

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	53.00%	Medium	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	88.00%	High	Increased	36200865

Zea mays

AC218148.2_FG008

Mitochondrion

rps7

332

CDS

Maizegdb database (www.maizegdb.org)

C-to-U

NA=>NA

**Experiment Details**
Genotype (Ecotype)	Allele	Treatment	Treatment Detail	Mutant Type	Phenotype	Tissue	Development Stage	Detection Method	Editing Frequency	Editing Extent	Mutant Effect	PMID
B73	WT	Control	Control	No mutant	Normal	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	68.00%	High	None	36200865
B73	bccp1-2	A Mu substitution	bccp1-2 contains a 1189 (A->C) mutation causing Lys->Gln and an 1192G->T mutation producing a stop codon, causing a truncation of 269 AAs at the C-terminus of AC218148.2_FG008	Recessive; Homozygous	The bccp1-2 mutant kernels at 12 DAP developed a barely visible embryo and an endosperm that was much smaller than that of the wild-type. Paraffin sectioning of the developing kernels at 12 DAP showed similar results. Reciprocal crosses between heterozygotes bccp1-1 and bccp1-2 produced the emp kernels at a 25% ratio, suggesting that AC218148.2_FG008 is the causal gene of the emp phenotype.	Kernel	12 DAP	Strand- and transcript-specific RNA-seq (STS-PCR-seq)	92.00%	High	Increased	36200865

Editing Factor:	bCCP1
Synonym:	bZIP and coiled-coil domain-containing PPR 1
Description:	bCCP1 interacts with PCW1 and the PPR-E protein Empty pericarp7 (EMP7); Two multiple organellar RNA editing factor (MORF) proteins, ZmMORF1 and ZmMORF8, interact with PCW1, EMP7, and bCCP1; C-to-U editing at the ccmFN-1553 site in maize required EMP7, bCCP1, and PCW1; PPR-E proteins function in RNA editing by recruiting the trans deaminase PCW1 and bCCP1, and MORF1/8 assist this recruitment through protein-protein interactions
Protein Family:	NA
Subclass:	NA
Physical Interaction:	PCW1; EMP7; ZmMORF1; ZmMORF8
Construct Structure:	bZIP-P-CC
Gene ID & Species:	AC218148.2_FG008 (Zea mays)
Edited Gene(s):	cob cox2 cox3 nad1 nad2 nad4L nad5 nad6 nad7 nad9 ccmB ccmFc ccmFn atp4 atp6 rps1 rps2A rps2B rps3 rps4 rps13 rpl16 matR atp8 atp9 cox1 nad4 mttB nad3 rps12 rps7
Editing Type(s):	C-to-U (360)
Publication(s):	[1] Maize PPR-E proteins mediate RNA C-to-U editing in mitochondria by recruiting the trans deaminase PCW1., The Plant cell, 2023. [PMID=36200865]

Summary

Editing Details